Page 1 | Search Results | Journal of Experimental Medicine

1-11 of 11

Sort by

Sort Order Select

Journal Articles

Human PIK3R1 mutations disrupt lymphocyte differentiation to cause activated PI3Kδ syndrome 2

J Exp Med (2023) 220 (6): e20221020.

https://doi.org/10.1084/jem.20221020

Published: 21 March 2023

View article titled, Human PIK3R1 mutations disrupt lymphocyte differentiation to cause activated PI3Kδ syndrome 2

Includes: Supplementary data

Journal Articles

Biochemically deleterious human NFKB1 variants underlie an autosomal dominant form of common variable immunodeficiency

J Exp Med (2021) 218 (11): e20210566.

https://doi.org/10.1084/jem.20210566

Published: 02 September 2021

View article titled, Biochemically deleterious human NFKB1 variants underlie an autosomal dominant form of common variable immunodeficiency

Includes: Supplementary data

Journal Articles

Impaired respiratory burst contributes to infections in PKCδ-deficient patients

J Exp Med (2021) 218 (9): e20210501.

https://doi.org/10.1084/jem.20210501

Published: 15 July 2021

View article titled, Impaired respiratory burst contributes to infections in PKCδ-deficient patients

Includes: Supplementary data

Journal Articles

Activated PI3Kδ breaches multiple B cell tolerance checkpoints and causes autoantibody production

J Exp Med (2019) 217 (2): e20191336.

https://doi.org/10.1084/jem.20191336

Published: 16 December 2019

View article titled, Activated PI3Kδ breaches multiple B cell tolerance checkpoints and causes autoantibody production

Includes: Supplementary data

Journal Articles

Germline-activating mutations in PIK3CD compromise B cell development and function

In Special Collection:

Inborn Errors of Immunity 2019

J Exp Med (2018) 215 (8): 2073–2095.

https://doi.org/10.1084/jem.20180010

Published: 17 July 2018

View article titled, Germline-activating mutations in PIK3CD compromise B cell development and function

Includes: Supplementary data

Journal Articles

Correction: Self-reactive VH4-34–expressing IgG B cells recognize commensal bacteria

Jean-Nicolas Schickel, Salomé Glauzy, Yen-Shing Ng, Nicolas Chamberlain, Christopher Massad, Isabelle Isnardi, Nathan Katz, Gulbu Uzel, Steven M. Holland, Capucine Picard, Anne Puel, Jean-Laurent Casanova, Eric Meffre

J Exp Med (2017) 214 (7): 2161.

https://doi.org/10.1084/jem.2016020105152017c

Published: 18 May 2017

View article titled, Correction: Self-reactive VH4-34–expressing IgG B cells recognize commensal bacteria

Journal Articles

Self-reactive VH4-34–expressing IgG B cells recognize commensal bacteria

J Exp Med (2017) 214 (7): 1991–2003.

https://doi.org/10.1084/jem.20160201

Published: 12 May 2017

View article titled, Self-reactive VH4-34–expressing IgG B cells recognize commensal bacteria

Includes: Supplementary data

Journal Articles

Unique and shared signaling pathways cooperate to regulate the differentiation of human CD4⁺ T cells into distinct effector subsets

J Exp Med (2016) 213 (8): 1589–1608.

https://doi.org/10.1084/jem.20151467

Published: 11 July 2016

View article titled, Unique and shared signaling pathways cooperate to regulate the differentiation of human CD4+ T cells into distinct effector subsets

Includes: Supplementary data

Journal Articles

STAT3 is a critical cell-intrinsic regulator of human unconventional T cell numbers and function

J Exp Med (2015) 212 (6): 855–864.

https://doi.org/10.1084/jem.20141992

Published: 04 May 2015

View article titled, STAT3 is a critical cell-intrinsic regulator of human unconventional T cell numbers and function

Journal Articles

Naive and memory human B cells have distinct requirements for STAT3 activation to differentiate into antibody-secreting plasma cells

J Exp Med (2013) 210 (12): 2739–2753.

https://doi.org/10.1084/jem.20130323

Published: 11 November 2013

View article titled, Naive and memory human B cells have distinct requirements for STAT3 activation to differentiate into antibody-secreting plasma cells

Includes: Supplementary data

Journal Articles

Loss-of-function mutations in the IL-21 receptor gene cause a primary immunodeficiency syndrome

J Exp Med (2013) 210 (3): 433–443.

https://doi.org/10.1084/jem.20111229

Published: 25 February 2013

View article titled, Loss-of-function mutations in the IL-21 receptor gene cause a primary immunodeficiency syndrome

Includes: Supplementary data

NARROW

Human PIK3R1 mutations disrupt lymphocyte differentiation to cause activated PI3Kδ syndrome 2

Biochemically deleterious human NFKB1 variants underlie an autosomal dominant form of common variable immunodeficiency

Impaired respiratory burst contributes to infections in PKCδ-deficient patients

Activated PI3Kδ breaches multiple B cell tolerance checkpoints and causes autoantibody production

Germline-activating mutations in PIK3CD compromise B cell development and function

Correction: Self-reactive VH4-34–expressing IgG B cells recognize commensal bacteria

Self-reactive VH4-34–expressing IgG B cells recognize commensal bacteria

Unique and shared signaling pathways cooperate to regulate the differentiation of human CD4⁺ T cells into distinct effector subsets

STAT3 is a critical cell-intrinsic regulator of human unconventional T cell numbers and function

Naive and memory human B cells have distinct requirements for STAT3 activation to differentiate into antibody-secreting plasma cells

Loss-of-function mutations in the IL-21 receptor gene cause a primary immunodeficiency syndrome

Email alerts

Email alerts

This Feature Is Available To Subscribers Only