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Journal Articles

SARS-CoV-2 infection and recovery in children: Distinct T cell responses in MIS-C compared to COVID-19

In Special Collection:

Journal-Based CME Activities with Journal of Experimental Medicine (JEM)

Ksenia Rybkina, Joseph N. Bell, Marissa C. Bradley, Teddy Wohlbold, Marika Scafuro, Wenzhao Meng, Rebecca C. Korenberg, Julia Davis-Porada, Brett R. Anderson, Rachel J. Weller, Joshua D. Milner, Anne Moscona, Matteo Porotto, Eline T. Luning Prak, Kalpana Pethe, Thomas J. Connors, Donna L. Farber

J Exp Med (2023) 220 (8): e20221518.

DOI: https://doi.org/10.1084/jem.20221518

Published: 03 May 2023

View article titled, SARS-CoV-2 infection and recovery in children: Distinct T cell responses in MIS-C compared to COVID-19

Includes: Supplementary data

Journal Articles

SARS-CoV-2–related MIS-C: A key to the viral and genetic causes of Kawasaki disease?

Vanessa Sancho-Shimizu, Petter Brodin, Aurélie Cobat, Catherine M. Biggs, Julie Toubiana, Carrie L. Lucas, Sarah E. Henrickson, Alexandre Belot, MIS-C@CHGE, Stuart G. Tangye, Joshua D. Milner, Michael Levin, Laurent Abel, Dusan Bogunovic, Jean-Laurent Casanova, Shen-Ying Zhang

J Exp Med (2021) 218 (6): e20210446.

DOI: https://doi.org/10.1084/jem.20210446

Published: 27 April 2021

View article titled, SARS-CoV-2–related MIS-C: A key to the viral and genetic causes of Kawasaki disease?

Includes: Supplementary data

Journal Articles

Inherited SLP76 deficiency in humans causes severe combined immunodeficiency, neutrophil and platelet defects

J Exp Med (2020) 218 (3): e20201062.

DOI: https://doi.org/10.1084/jem.20201062

Published: 24 November 2020

View article titled, Inherited SLP76 deficiency in humans causes severe combined immunodeficiency, neutrophil and platelet defects

Includes: Supplementary data

Journal Articles

Correction: Dominant-negative mutations in human IL6ST underlie hyper-IgE syndrome

Vivien Béziat, Simon J. Tavernier, Yin-Huai Chen, Cindy S. Ma, Marie Materna, Arian Laurence, Jens Staal, Dominik Aschenbrenner, Lisa Roels, Lisa Worley, Kathleen Claes, Lisa Gartner, Lisa A. Kohn, Marieke De Bruyne, Klaus Schmitz-Abe, Louis-Marie Charbonnier, Sevgi Keles, Justine Nammour, Natasha Vladikine, Majistor Raj Luxman Maglorius Renkilaraj, Yoann Seeleuthner, Mélanie Migaud, Jérémie Rosain, Mohamed Jeljeli, Bertrand Boisson, Eva Van Braeckel, Jill A. Rosenfeld, Hongzheng Dai, Lindsay C. Burrage, David R. Murdock, Bart N. Lambrecht, Véronique Avettand-Fenoel, Tiphanie P. Vogel, Undiagnosed Diseases Network, Charles R. Esther, Jr., Sule Haskologlu, Figen Dogu, Peter Ciznar, David Boutboul, Marie Ouachée-Chardin, Jean Amourette, Marie-Noëlle Lebras, Clément Gauvain, Colas Tcherakian, Aydan Ikinciogullari, Rudi Beyaert, Laurent Abel, Joshua D. Milner, Bodo Grimbacher, Louis-Jean Couderc, Manish J. Butte, Alexandra F. Freeman, Émilie Catherinot, Claire Fieschi, Talal A. Chatila, Stuart G. Tangye, Holm H. Uhlig, Filomeen Haerynck, Jean-Laurent Casanova, Anne Puel

J Exp Med (2020) 217 (7): e2019180405272020c.

DOI: https://doi.org/10.1084/jem.2019180405272020c

Published: 09 June 2020

View article titled, Correction: Dominant-negative mutations in human IL6ST underlie hyper-IgE syndrome

Journal Articles

Dominant-negative mutations in human IL6ST underlie hyper-IgE syndrome

In Special Collection:

Cytokines Collection 2020

J Exp Med (2020) 217 (6): e20191804.

DOI: https://doi.org/10.1084/jem.20191804

Published: 24 March 2020

View article titled, Dominant-negative mutations in human IL6ST underlie hyper-IgE syndrome

Includes: Supplementary data

Journal Articles

Impact of naturally forming human α/β-tryptase heterotetramers in the pathogenesis of hereditary α-tryptasemia

Quang T. Le, Jonathan J. Lyons, Andrea N. Naranjo, Ana Olivera, Robert A. Lazarus, Dean D. Metcalfe, Joshua D. Milner, Lawrence B. Schwartz

J Exp Med (2019) 216 (10): 2348–2361.

DOI: https://doi.org/10.1084/jem.20190701

Published: 23 July 2019

View article titled, Impact of naturally forming human α/β-tryptase heterotetramers in the pathogenesis of hereditary α-tryptasemia

Includes: Supplementary data

Journal Articles

Loss of the interleukin-6 receptor causes immunodeficiency, atopy, and abnormal inflammatory responses

In Special Collection:

2019: The Year in Experimental Medicine

J Exp Med (2019) 216 (9): 1986–1998.

DOI: https://doi.org/10.1084/jem.20190344

Published: 24 June 2019

View article titled, Loss of the interleukin-6 receptor causes immunodeficiency, atopy, and abnormal inflammatory responses

Includes: Supplementary data

Journal Articles

Primary atopic disorders

In Special Collection:

Monogenic Auto-Inflammation and Auto-Immunity in Humans 2018

Jonathan J. Lyons, Joshua D. Milner

J Exp Med (2018) 215 (4): 1009–1022.

DOI: https://doi.org/10.1084/jem.20172306

Published: 16 March 2018

View article titled, Primary atopic disorders

Journal Articles

A biallelic mutation in IL6ST encoding the GP130 co-receptor causes immunodeficiency and craniosynostosis

J Exp Med (2017) 214 (9): 2547–2562.

DOI: https://doi.org/10.1084/jem.20161810

Published: 26 July 2017

View article titled, A biallelic mutation in IL6ST encoding the GP130 co-receptor causes immunodeficiency and craniosynostosis

Includes: Supplementary data

Journal Articles

PD-L1 up-regulation restrains Th17 cell differentiation in STAT3 loss- and STAT1 gain-of-function patients

In Special Collection:

Inborn Errors of Immunity 2019

Yuan Zhang, Chi A. Ma, Monica G. Lawrence, Timothy J. Break, Michael P. O’Connell, Jonathan J. Lyons, Diego B. López, John S. Barber, Yongge Zhao, Daniel L. Barber, Alexandra F. Freeman, Steven M. Holland, Michail S. Lionakis, Joshua D. Milner

J Exp Med (2017) 214 (9): 2523–2533.

DOI: https://doi.org/10.1084/jem.20161427

Published: 14 July 2017

View article titled, PD-L1 up-regulation restrains Th17 cell differentiation in STAT3 loss- and STAT1 gain-of-function patients

Includes: Supplementary data

Journal Articles

Loss-of-function mutations in the IL-21 receptor gene cause a primary immunodeficiency syndrome

J Exp Med (2013) 210 (3): 433–443.

DOI: https://doi.org/10.1084/jem.20111229

Published: 25 February 2013

View article titled, Loss-of-function mutations in the IL-21 receptor gene cause a primary immunodeficiency syndrome

Includes: Supplementary data

NARROW

SARS-CoV-2 infection and recovery in children: Distinct T cell responses in MIS-C compared to COVID-19

SARS-CoV-2–related MIS-C: A key to the viral and genetic causes of Kawasaki disease?

Inherited SLP76 deficiency in humans causes severe combined immunodeficiency, neutrophil and platelet defects

Correction: Dominant-negative mutations in human IL6ST underlie hyper-IgE syndrome

Dominant-negative mutations in human IL6ST underlie hyper-IgE syndrome

Impact of naturally forming human α/β-tryptase heterotetramers in the pathogenesis of hereditary α-tryptasemia

Loss of the interleukin-6 receptor causes immunodeficiency, atopy, and abnormal inflammatory responses

Primary atopic disorders

A biallelic mutation in IL6ST encoding the GP130 co-receptor causes immunodeficiency and craniosynostosis

PD-L1 up-regulation restrains Th17 cell differentiation in STAT3 loss- and STAT1 gain-of-function patients

Loss-of-function mutations in the IL-21 receptor gene cause a primary immunodeficiency syndrome

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