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Researchers describe how environmental signals trigger wound repair in zebrafish.

People & Ideas

Bergmann studies stomatal development to discover how plant cells parse polarity and fate.


Active recruitment of Aurora B and its counteracting phosphatase PP2A–B56 to the anaphase spindle is promoted by the kinesins MKlp2 and KIF4A, respectively.

Starvation triggers relocation of membranes of the secretory compartments and endosomes to create a compartment called CUPS, which may be involved in processing and secretion of proteins that cannot enter the ER–Golgi pathway.

DisAp is a novel kinetodesmal fiber component that is essential for force-dependent fiber elongation and the alignment of basal body orientation in multiciliary arrays.


Chromatin compaction mediated by CHD3.1 must be counteracted by ACF1–SNF2H and RNF20 in order to allow DNA double-strand break repair in heterochromatin of postreplicative cells.

Interactions between ankyrin-B and both dynactin and phosphatidylinositol 3-phosphate lipids promote fast axonal transport of organelles.

Thyroid hormone promotes slow-to-fast muscle fiber type conversion by inducing miR-133a1 and thereby repressing the expression of the slow muscle determinant TEAD1.

After wounding in zebrafish, osmolarity differences between the interstitial fluid and the external environment trigger ATP release that initiates rapid wound closure through long-range activation of basal epithelial cell motility.

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