Issues
1 August 2008
-
Cover Image
Cover Image
Cover picture: Absense of dystrophin in skeletal muscle cells causes disruption in localization and kinetic properties of Nav1.4 ion channels. (Top) Images of Nav1.4 immunofluorescence in single C57BL/6J (left) and mdx5cv (right) fibers show disruption in a longitudinally striated pattern in mdx. (Bottom) Representative traces of cell-attached Na+ currents in control (left) and mdx (right) fibers show increased activity and prolonged inactivation in mdx cells (see article by Hirn et al., 199–208).
- PDF Icon PDF LinkTable of Contents
- PDF Icon PDF LinkEditorial Board
ISSN 0022-1295
EISSN 1540-7748
In this Issue
Article
Tutorial
Letter to the Editor
Email alerts
Most Read
Voltage-Gated Na Channels
Drugs exhibit diverse binding modes and access routes in the Nav1.5 cardiac sodium channel pore
Mechanotransduction by Membrane Proteins
Mechanosensitive channel MscS is critical for termination of the bacterial hypoosmotic permeability response
Advertisement
