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1 August 2008
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Cover Image
Cover Image
Cover picture: Absense of dystrophin in skeletal muscle cells causes disruption in localization and kinetic properties of Nav1.4 ion channels. (Top) Images of Nav1.4 immunofluorescence in single C57BL/6J (left) and mdx5cv (right) fibers show disruption in a longitudinally striated pattern in mdx. (Bottom) Representative traces of cell-attached Na+ currents in control (left) and mdx (right) fibers show increased activity and prolonged inactivation in mdx cells (see article by Hirn et al., 199–208).
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ISSN 0022-1295
EISSN 1540-7748
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Excitation–Contraction Coupling
Orai1 as a potential “fits-all approach” therapeutic target for the treatment of DMD
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