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D Mason
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Journal Articles

Structural and genetic basis of HIV-1 envelope V2 apex recognition by rhesus broadly neutralizing antibodies

Ryan S. Roark, Rumi Habib, Jason Gorman, Hui Li, Andrew Jesse Connell, Mattia Bonsignori, Yicheng Guo, Michael P. Hogarty, Adam S. Olia, Kirsten J. Sowers, Baoshan Zhang, Frederic Bibollet-Ruche, Tatsiana Bylund, Sean Callaghan, John W. Carey, Gabriele Cerutti, Darcy R. Harris, Wanting He, Emily Lewis, Tracy Liu, Rosemarie D. Mason, Yujie Qiao, Younghoon Park, Juliette M. Rando, Ajay Singh, Jeremy J. Wolff, Q. Paula Lei, Mark K. Louder, Raiees Andrabi, Nicole A. Doria-Rose, Kevin O. Saunders, Michael S. Seaman, Barton F. Haynes, Daniel W. Kulp, John R. Mascola, Mario Roederer, Theodore C. Pierson, Zizhang Sheng, Beatrice H. Hahn, George M. Shaw, Peter D. Kwong, Lawrence Shapiro
Journal: Journal of Experimental Medicine
J Exp Med (2025) 222 (10): e20250638.
https://doi.org/10.1084/jem.20250638
Published: 18 August 2025
View article titled, Structural and genetic basis of HIV-1 envelope V2 apex recognition by rhesus broadly neutralizing antibodies
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Includes: Supplementary data
Journal Articles

Megakaryocytes possess a functional intrinsic apoptosis pathway that must be restrained to survive and produce platelets

Emma C. Josefsson, Chloé James, Katya J. Henley, Marlyse A. Debrincat, Kelly L. Rogers, Mark R. Dowling, Michael J. White, Elizabeth A. Kruse, Rachael M. Lane, Sarah Ellis, Paquita Nurden, Kylie D. Mason, Lorraine A. O’Reilly, Andrew W. Roberts, Donald Metcalf, David C.S. Huang, Benjamin T. Kile
Journal: Journal of Experimental Medicine
J Exp Med (2011) 208 (10): 2017–2031.
https://doi.org/10.1084/jem.20110750
Published: 12 September 2011
View article titled, Megakaryocytes possess a functional intrinsic apoptosis pathway that must be restrained to survive and produce platelets
Open the PDF for Megakaryocytes possess a functional intrinsic apoptosis pathway that must be restrained to survive and produce platelets in another window
Includes: Supplementary data
Journal Articles

The role of B cells in the programming of T cells for IL-4 synthesis.

D Mason
Journal: Journal of Experimental Medicine
J Exp Med (1996) 183 (3): 717–719.
https://doi.org/10.1084/jem.183.3.717
Published: 01 March 1996
View article titled, The role of B cells in the programming of T cells for IL-4 synthesis.
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Journal Articles

Prevention of experimental allergic encephalomyelitis in rats by targeting autoantigen to B cells: evidence that the protective mechanism depends on changes in the cytokine response and migratory properties of the autoantigen-specific T cells.

A Saoudi, S Simmonds, I Huitinga, D Mason
Journal: Journal of Experimental Medicine
J Exp Med (1995) 182 (2): 335–344.
https://doi.org/10.1084/jem.182.2.335
Published: 01 August 1995
View article titled, Prevention of experimental allergic encephalomyelitis in rats by targeting autoantigen to B cells: evidence that the protective mechanism depends on changes in the cytokine response and migratory properties of the autoantigen-specific T cells.
Open the PDF for Prevention of experimental allergic encephalomyelitis in rats by targeting autoantigen to B cells: evidence that the protective mechanism depends on changes in the cytokine response and migratory properties of the autoantigen-specific T cells. in another window
Journal Articles

Activation of CD4+ T cells in the presence of a nondepleting monoclonal antibody to CD4 induces a Th2-type response in vitro.

P Stumbles, D Mason
Journal: Journal of Experimental Medicine
J Exp Med (1995) 182 (1): 5–13.
https://doi.org/10.1084/jem.182.1.5
Published: 01 July 1995
View article titled, Activation of CD4+ T cells in the presence of a nondepleting monoclonal antibody to CD4 induces a Th2-type response in vitro.
Open the PDF for Activation of CD4+ T cells in the presence of a nondepleting monoclonal antibody to CD4 induces a Th2-type response in vitro. in another window
Journal Articles

Evidence that the T cell repertoire of normal rats contains cells with the potential to cause diabetes. Characterization of the CD4+ T cell subset that inhibits this autoimmune potential.

D Fowell, D Mason
Journal: Journal of Experimental Medicine
J Exp Med (1993) 177 (3): 627–636.
https://doi.org/10.1084/jem.177.3.627
Published: 01 March 1993
View article titled, Evidence that the T cell repertoire of normal rats contains cells with the potential to cause diabetes. Characterization of the CD4+ T cell subset that inhibits this autoimmune potential.
Open the PDF for Evidence that the T cell repertoire of normal rats contains cells with the potential to cause diabetes. Characterization of the CD4+ T cell subset that inhibits this autoimmune potential. in another window
Journal Articles

Glucocorticoids induce the expression of CD8 alpha chains on concanavalin A-activated rat CD4+ T cells: induction is inhibited by rat recombinant interleukin 4.

F Ramirez, A J McKnight, A Silva, D Mason
Journal: Journal of Experimental Medicine
J Exp Med (1992) 176 (6): 1551–1559.
https://doi.org/10.1084/jem.176.6.1551
Published: 01 December 1992
View article titled, Glucocorticoids induce the expression of CD8 alpha chains on concanavalin A-activated rat CD4+ T cells: induction is inhibited by rat recombinant interleukin 4.
Open the PDF for Glucocorticoids induce the expression of CD8 alpha chains on concanavalin A-activated rat CD4+ T cells: induction is inhibited by rat recombinant interleukin 4. in another window
Journal Articles

OX-22high CD4+ T cells induce wasting disease with multiple organ pathology: prevention by the OX-22low subset.

F Powrie, D Mason
Journal: Journal of Experimental Medicine
J Exp Med (1990) 172 (6): 1701–1708.
https://doi.org/10.1084/jem.172.6.1701
Published: 01 December 1990
View article titled, OX-22high CD4+ T cells induce wasting disease with multiple organ pathology: prevention by the OX-22low subset.
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Journal Articles

The MRC OX-22- CD4+ T cells that help B cells in secondary immune responses derive from naive precursors with the MRC OX-22+ CD4+ phenotype.

F Powrie, D Mason
Journal: Journal of Experimental Medicine
J Exp Med (1989) 169 (3): 653–662.
https://doi.org/10.1084/jem.169.3.653
Published: 01 March 1989
View article titled, The MRC OX-22- CD4+ T cells that help B cells in secondary immune responses derive from naive precursors with the MRC OX-22+ CD4+ phenotype.
Open the PDF for The MRC OX-22- CD4+ T cells that help B cells in secondary immune responses derive from naive precursors with the MRC OX-22+ CD4+ phenotype. in another window
Journal Articles

Experimental allergic encephalomyelitis in the absence of a classical delayed-type hypersensitivity reaction. Severe paralytic disease correlates with the presence of interleukin 2 receptor-positive cells infiltrating the central nervous system.

J Sedgwick, S Brostoff, D Mason
Journal: Journal of Experimental Medicine
J Exp Med (1987) 165 (4): 1058–1075.
https://doi.org/10.1084/jem.165.4.1058
Published: 01 April 1987
View article titled, Experimental allergic encephalomyelitis in the absence of a classical delayed-type hypersensitivity reaction. Severe paralytic disease correlates with the presence of interleukin 2 receptor-positive cells infiltrating the central nervous system.
Open the PDF for Experimental allergic encephalomyelitis in the absence of a classical delayed-type hypersensitivity reaction. Severe paralytic disease correlates with the presence of interleukin 2 receptor-positive cells infiltrating the central nervous system. in another window
Journal Articles

T cells that help B cell responses to soluble antigen are distinguishable from those producing interleukin 2 on mitogenic or allogeneic stimulation.

R P Arthur, D Mason
Journal: Journal of Experimental Medicine
J Exp Med (1986) 163 (4): 774–786.
https://doi.org/10.1084/jem.163.4.774
Published: 01 April 1986
View article titled, T cells that help B cell responses to soluble antigen are distinguishable from those producing interleukin 2 on mitogenic or allogeneic stimulation.
Open the PDF for T cells that help B cell responses to soluble antigen are distinguishable from those producing interleukin 2 on mitogenic or allogeneic stimulation. in another window