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Journal Articles

H-2M3a violates the paradigm for major histocompatibility complex class I peptide binding.

J M Vyas, J R Rodgers, R R Rich

J Exp Med (1995) 181 (5): 1817–1825.

https://doi.org/10.1084/jem.181.5.1817

Published: 01 May 1995

View article titled, H-2M3a violates the paradigm for major histocompatibility complex class I peptide binding.

Journal Articles

Availability of endogenous peptides limits expression of an M3a-Ld major histocompatibility complex class I chimera.

J M Vyas, R R Rich, D D Howell, S M Shawar, J R Rodgers

J Exp Med (1994) 179 (1): 155–165.

https://doi.org/10.1084/jem.179.1.155

Published: 01 January 1994

View article titled, Availability of endogenous peptides limits expression of an M3a-Ld major histocompatibility complex class I chimera.

Journal Articles

Localization of a site on bacterial superantigens that determines T cell receptor beta chain specificity.

J A Mollick, R L McMasters, D Grossman, R R Rich

J Exp Med (1993) 177 (2): 283–293.

https://doi.org/10.1084/jem.177.2.283

Published: 01 February 1993

View article titled, Localization of a site on bacterial superantigens that determines T cell receptor beta chain specificity.

Journal Articles

Specialized functions of major histocompatibility complex class I molecules. II. Hmt binds N-formylated peptides of mitochondrial and prokaryotic origin.

S M Shawar, J M Vyas, J R Rodgers, R G Cook, R R Rich

J Exp Med (1991) 174 (4): 941–944.

https://doi.org/10.1084/jem.174.4.941

Published: 01 October 1991

View article titled, Specialized functions of major histocompatibility complex class I molecules. II. Hmt binds N-formylated peptides of mitochondrial and prokaryotic origin.

Journal Articles

Dissociation of the stimulatory activities of staphylococcal enterotoxins for T cells and monocytes.

D Grossman, R G Cook, J T Sparrow, J A Mollick, R R Rich

J Exp Med (1990) 172 (6): 1831–1841.

https://doi.org/10.1084/jem.172.6.1831

Published: 01 December 1990

View article titled, Dissociation of the stimulatory activities of staphylococcal enterotoxins for T cells and monocytes.

Journal Articles

Specialized functions of MHC class I molecules. I. An N-formyl peptide receptor is required for construction of the class I antigen Mta.

S M Shawar, R G Cook, J R Rodgers, R R Rich

J Exp Med (1990) 171 (3): 897–912.

https://doi.org/10.1084/jem.171.3.897

Published: 01 March 1990

View article titled, Specialized functions of MHC class I molecules. I. An N-formyl peptide receptor is required for construction of the class I antigen Mta.

Journal Articles

T suppressor cell growth factor and anti-CD3 antibodies stimulate reciprocal subsets of T lymphocytes.

E J Fox, D E Lewis, K P Deemer, M N ElMasry, R R Rich

J Exp Med (1987) 166 (2): 404–418.

https://doi.org/10.1084/jem.166.2.404

Published: 01 August 1987

View article titled, T suppressor cell growth factor and anti-CD3 antibodies stimulate reciprocal subsets of T lymphocytes.

Journal Articles

Maternally transmitted antigens are codominantly expressed by mouse cells containing two kinds of mitochondrial DNA.

J R Rodgers, R Smith, 3rd, R R Rich

J Exp Med (1987) 165 (2): 560–565.

https://doi.org/10.1084/jem.165.2.560

Published: 01 February 1987

View article titled, Maternally transmitted antigens are codominantly expressed by mouse cells containing two kinds of mitochondrial DNA.

Journal Articles

Differences in maternal lineages of New Zealand Black mice defined by restriction endonuclease analysis of mitochondrial DNA and by expression of maternally transmitted antigen.

M M Huston, R Smith, 3rd, D P Huston, R R Rich

J Exp Med (1983) 157 (6): 2154–2159.

https://doi.org/10.1084/jem.157.6.2154

Published: 01 June 1983

View article titled, Differences in maternal lineages of New Zealand Black mice defined by restriction endonuclease analysis of mitochondrial DNA and by expression of maternally transmitted antigen.

Journal Articles

Primary cell-mediated lympholysis response to a maternally transmitted antigen.

R Smith, 3rd, D P Huston, R R Rich

J Exp Med (1982) 156 (6): 1866–1871.

https://doi.org/10.1084/jem.156.6.1866

Published: 01 December 1982

View article titled, Primary cell-mediated lympholysis response to a maternally transmitted antigen.

Journal Articles

Antigen presentation to human T lymphocytes. I. Different requirements for stimulation by hapten-modified cells vs. cell sonicates.

S L Abramson, J M Puck, R R Rich

J Exp Med (1981) 154 (4): 1005–1015.

https://doi.org/10.1084/jem.154.4.1005

Published: 01 October 1981

View article titled, Antigen presentation to human T lymphocytes. I. Different requirements for stimulation by hapten-modified cells vs. cell sonicates.

Journal Articles

Primary in vitro cytotoxic response of NZB spleen cells to Qa-1b-associated antigenic determinants.

R R Rich, D A Sedberry, D L Kastner, L Chu

J Exp Med (1979) 150 (6): 1555–1560.

https://doi.org/10.1084/jem.150.6.1555

Published: 01 December 1979

View article titled, Primary in vitro cytotoxic response of NZB spleen cells to Qa-1b-associated antigenic determinants.

Journal Articles

Regulatory mechanisms in cell-mediated immune responses. VII. Presence of I-C subregion determinants on mixed leukocyte reaction suppressor factor.

S S Rich, C S David, R R Rich

J Exp Med (1979) 149 (1): 114–126.

https://doi.org/10.1084/jem.149.1.114

Published: 01 January 1979

View article titled, Regulatory mechanisms in cell-mediated immune responses. VII. Presence of I-C subregion determinants on mixed leukocyte reaction suppressor factor.

Journal Articles

Human immune responses to hapten-conjugated cells. I. Primary and secondary proliferative responses in vitro.

M F Seldin, R R Rich

J Exp Med (1978) 147 (6): 1671–1683.

https://doi.org/10.1084/jem.147.6.1671

Published: 01 June 1978

View article titled, Human immune responses to hapten-conjugated cells. I. Primary and secondary proliferative responses in vitro.

Journal Articles

Regulatory mechanisms in cell-mediated immune responses. V. H-2 homology requirements for the production of a minor locus-induced suppressor factor.

D L Kastner, R R Rich, L Chu, S S Rich

J Exp Med (1977) 146 (4): 1152–1157.

https://doi.org/10.1084/jem.146.4.1152

Published: 01 October 1977

View article titled, Regulatory mechanisms in cell-mediated immune responses. V. H-2 homology requirements for the production of a minor locus-induced suppressor factor.

Journal Articles

Regulatory mechanisms in cell-mediated immune responses. IV. Expression of a receptor for mixed lymphocyte reaction suppressor factor on activated T lymphocytes.

S S Rich, R R Rich

J Exp Med (1976) 144 (5): 1214–1226.

https://doi.org/10.1084/jem.144.5.1214

Published: 01 November 1976

View article titled, Regulatory mechanisms in cell-mediated immune responses. IV. Expression of a receptor for mixed lymphocyte reaction suppressor factor on activated T lymphocytes.

Journal Articles

Regulatory mechanisms in cell-mediated immune responses. III. I-region control of suppressor cell interaction with responder cells in mixed lymphocyte reactions.

S S Rich, R R Rich

J Exp Med (1976) 143 (3): 672–677.

https://doi.org/10.1084/jem.143.3.672

Published: 01 March 1976

View article titled, Regulatory mechanisms in cell-mediated immune responses. III. I-region control of suppressor cell interaction with responder cells in mixed lymphocyte reactions.

Journal Articles

Regulatory mechanisms in cell-mediated immune responses. II. A genetically restricted suppressor of mixed lymphocyte reactions released by alloantigen-activated spleen cells.

S S Rich, R R Rich

J Exp Med (1975) 142 (6): 1391–1402.

https://doi.org/10.1084/jem.142.6.1391

Published: 01 December 1975

View article titled, Regulatory mechanisms in cell-mediated immune responses. II. A genetically restricted suppressor of mixed lymphocyte reactions released by alloantigen-activated spleen cells.

NARROW

H-2M3a violates the paradigm for major histocompatibility complex class I peptide binding.

Availability of endogenous peptides limits expression of an M3a-Ld major histocompatibility complex class I chimera.

Localization of a site on bacterial superantigens that determines T cell receptor beta chain specificity.

Specialized functions of major histocompatibility complex class I molecules. II. Hmt binds N-formylated peptides of mitochondrial and prokaryotic origin.

Dissociation of the stimulatory activities of staphylococcal enterotoxins for T cells and monocytes.

Specialized functions of MHC class I molecules. I. An N-formyl peptide receptor is required for construction of the class I antigen Mta.

T suppressor cell growth factor and anti-CD3 antibodies stimulate reciprocal subsets of T lymphocytes.

Maternally transmitted antigens are codominantly expressed by mouse cells containing two kinds of mitochondrial DNA.

Differences in maternal lineages of New Zealand Black mice defined by restriction endonuclease analysis of mitochondrial DNA and by expression of maternally transmitted antigen.

Primary cell-mediated lympholysis response to a maternally transmitted antigen.

Antigen presentation to human T lymphocytes. I. Different requirements for stimulation by hapten-modified cells vs. cell sonicates.

Primary in vitro cytotoxic response of NZB spleen cells to Qa-1b-associated antigenic determinants.

Regulatory mechanisms in cell-mediated immune responses. VII. Presence of I-C subregion determinants on mixed leukocyte reaction suppressor factor.

Human immune responses to hapten-conjugated cells. I. Primary and secondary proliferative responses in vitro.

Regulatory mechanisms in cell-mediated immune responses. V. H-2 homology requirements for the production of a minor locus-induced suppressor factor.

Regulatory mechanisms in cell-mediated immune responses. IV. Expression of a receptor for mixed lymphocyte reaction suppressor factor on activated T lymphocytes.

Regulatory mechanisms in cell-mediated immune responses. III. I-region control of suppressor cell interaction with responder cells in mixed lymphocyte reactions.

Regulatory mechanisms in cell-mediated immune responses. II. A genetically restricted suppressor of mixed lymphocyte reactions released by alloantigen-activated spleen cells.

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