Page 1 | Search Results | Journal of Experimental Medicine

Skip Nav Destination

1-5 of 5

Sort by

Sort Order Select

Journal Articles

The relative contribution of the CD28 and gp39 costimulatory pathways in the clonal expansion and pathogenic acquisition of self-reactive T cells.

N D Griggs, S S Agersborg, R J Noelle, J A Ledbetter, P S Linsley, K S Tung

J Exp Med (1996) 183 (3): 801–810.

https://doi.org/10.1084/jem.183.3.801

Published: 01 March 1996

View articletitled, The relative contribution of the CD28 and gp39 costimulatory pathways in the clonal expansion and pathogenic acquisition of self-reactive T cells.

Journal Articles

An essential role for gp39, the ligand for CD40, in thymic selection.

T M Foy, D M Page, T J Waldschmidt, A Schoneveld, J D Laman, S R Masters, L Tygrett, J A Ledbetter, A Aruffo, E Claassen, J C Xu, R A Flavell, S Oehen, S M Hedrick, R J Noelle

J Exp Med (1995) 182 (5): 1377–1388.

https://doi.org/10.1084/jem.182.5.1377

Published: 01 November 1995

View articletitled, An essential role for gp39, the ligand for CD40, in thymic selection.

Journal Articles

gp39-CD40 interactions are essential for germinal center formation and the development of B cell memory.

T M Foy, J D Laman, J A Ledbetter, A Aruffo, E Claassen, R J Noelle

J Exp Med (1994) 180 (1): 157–163.

https://doi.org/10.1084/jem.180.1.157

Published: 01 July 1994

View articletitled, gp39-CD40 interactions are essential for germinal center formation and the development of B cell memory.

Journal Articles

In vivo CD40-gp39 interactions are essential for thymus-dependent humoral immunity. I. In vivo expression of CD40 ligand, cytokines, and antibody production delineates sites of cognate T-B cell interactions.

A J Van den Eertwegh, R J Noelle, M Roy, D M Shepherd, A Aruffo, J A Ledbetter, W J Boersma, E Claassen

J Exp Med (1993) 178 (5): 1555–1565.

https://doi.org/10.1084/jem.178.5.1555

Published: 01 November 1993

View articletitled, In vivo CD40-gp39 interactions are essential for thymus-dependent humoral immunity. I. In vivo expression of CD40 ligand, cytokines, and antibody production delineates sites of cognate T-B cell interactions.

Journal Articles

In vivo CD40-gp39 interactions are essential for thymus-dependent humoral immunity. II. Prolonged suppression of the humoral immune response by an antibody to the ligand for CD40, gp39.

T M Foy, D M Shepherd, F H Durie, A Aruffo, J A Ledbetter, R J Noelle

J Exp Med (1993) 178 (5): 1567–1575.

https://doi.org/10.1084/jem.178.5.1567

Published: 01 November 1993

View articletitled, In vivo CD40-gp39 interactions are essential for thymus-dependent humoral immunity. II. Prolonged suppression of the humoral immune response by an antibody to the ligand for CD40, gp39.

Update search

NARROW

Format

Journal

Article Type

Date

The relative contribution of the CD28 and gp39 costimulatory pathways in the clonal expansion and pathogenic acquisition of self-reactive T cells.

An essential role for gp39, the ligand for CD40, in thymic selection.

gp39-CD40 interactions are essential for germinal center formation and the development of B cell memory.

In vivo CD40-gp39 interactions are essential for thymus-dependent humoral immunity. I. In vivo expression of CD40 ligand, cytokines, and antibody production delineates sites of cognate T-B cell interactions.

In vivo CD40-gp39 interactions are essential for thymus-dependent humoral immunity. II. Prolonged suppression of the humoral immune response by an antibody to the ligand for CD40, gp39.

Email alerts

Update search

The relative contribution of the CD28 and gp39 costimulatory pathways in the clonal expansion and pathogenic acquisition of self-reactive T cells.

An essential role for gp39, the ligand for CD40, in thymic selection.

gp39-CD40 interactions are essential for germinal center formation and the development of B cell memory.

In vivo CD40-gp39 interactions are essential for thymus-dependent humoral immunity. I. In vivo expression of CD40 ligand, cytokines, and antibody production delineates sites of cognate T-B cell interactions.

In vivo CD40-gp39 interactions are essential for thymus-dependent humoral immunity. II. Prolonged suppression of the humoral immune response by an antibody to the ligand for CD40, gp39.

Email alerts

This Feature Is Available To Subscribers Only