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Synergistic cooperation and crosstalk between MYD88^L265P and mutations that dysregulate CD79B and surface IgM

James Q. Wang, Yogesh S. Jeelall, Peter Humburg, Emma L. Batchelor, Sarp M. Kaya, Hee Min Yoo, Christopher C. Goodnow, Keisuke Horikawa

J Exp Med (2017) 214 (9): 2759–2776.

https://doi.org/10.1084/jem.20161454

Published: 12 July 2017

View articletitled, Synergistic cooperation and crosstalk between MYD88L265P and mutations that dysregulate CD79B and surface IgM

Includes: Supplementary data

Journal Articles

Consequences of the recurrent MYD88^L265P somatic mutation for B cell tolerance

James Q. Wang, Yogesh S. Jeelall, Bruce Beutler, Keisuke Horikawa, Christopher C. Goodnow

J Exp Med (2014) 211 (3): 413–426.

https://doi.org/10.1084/jem.20131424

Published: 17 February 2014

View articletitled, Consequences of the recurrent MYD88L265P somatic mutation for B cell tolerance

Journal Articles

B cell survival, surface BCR and BAFFR expression, CD74 metabolism, and CD8⁻ dendritic cells require the intramembrane endopeptidase SPPL2A

J Exp Med (2012) 210 (1): 31–40.

https://doi.org/10.1084/jem.20121076

Published: 24 December 2012

View articletitled, B cell survival, surface BCR and BAFFR expression, CD74 metabolism, and CD8− dendritic cells require the intramembrane endopeptidase SPPL2A

Journal Articles

Human lymphoma mutations reveal CARD11 as the switch between self-antigen–induced B cell death or proliferation and autoantibody production

Yogesh S. Jeelall, James Q. Wang, Hsei-Di Law, Heather Domaschenz, Herman K.H. Fung, Axel Kallies, Stephen L. Nutt, Christopher C. Goodnow, Keisuke Horikawa

J Exp Med (2012) 209 (11): 1907–1917.

https://doi.org/10.1084/jem.20112744

Published: 01 October 2012

View articletitled, Human lymphoma mutations reveal CARD11 as the switch between self-antigen–induced B cell death or proliferation and autoantibody production

Journal Articles

CD83 increases MHC II and CD86 on dendritic cells by opposing IL-10–driven MARCH1-mediated ubiquitination and degradation

Lina E. Tze, Keisuke Horikawa, Heather Domaschenz, Debbie R. Howard, Carla M. Roots, Robert J. Rigby, David A. Way, Mari Ohmura-Hoshino, Satoshi Ishido, Christopher E. Andoniou, Mariapia A. Degli-Esposti, Christopher C. Goodnow

J Exp Med (2011) 208 (1): 149–165.

https://doi.org/10.1084/jem.20092203

Published: 10 January 2011

View articletitled, CD83 increases MHC II and CD86 on dendritic cells by opposing IL-10–driven MARCH1-mediated ubiquitination and degradation

Includes: Supplementary data

Journal Articles

Enhancement and suppression of signaling by the conserved tail of IgG memory–type B cell antigen receptors

Keisuke Horikawa, Stephen W. Martin, Sarah L. Pogue, Karlee Silver, Kaiman Peng, Kiyoshi Takatsu, Christopher C. Goodnow

J Exp Med (2007) 204 (4): 759–769.

https://doi.org/10.1084/jem.20061923

Published: 09 April 2007

View articletitled, Enhancement and suppression of signaling by the conserved tail of IgG memory–type B cell antigen receptors

Includes: Supplementary data

Journal Articles

A Double-Edged Kinase Lyn: A Positive and Negative Regulator for Antigen Receptor–mediated Signals

Hirofumi Nishizumi, Keisuke Horikawa, Irena Mlinaric-Rascan, Tadashi Yamamoto

J Exp Med (1998) 187 (8): 1343–1348.

https://doi.org/10.1084/jem.187.8.1343

Published: 20 April 1998

View articletitled, A Double-Edged Kinase Lyn: A Positive and Negative Regulator for Antigen Receptor–mediated Signals

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Synergistic cooperation and crosstalk between MYD88^L265P and mutations that dysregulate CD79B and surface IgM

Consequences of the recurrent MYD88^L265P somatic mutation for B cell tolerance

B cell survival, surface BCR and BAFFR expression, CD74 metabolism, and CD8⁻ dendritic cells require the intramembrane endopeptidase SPPL2A

Human lymphoma mutations reveal CARD11 as the switch between self-antigen–induced B cell death or proliferation and autoantibody production

CD83 increases MHC II and CD86 on dendritic cells by opposing IL-10–driven MARCH1-mediated ubiquitination and degradation

Enhancement and suppression of signaling by the conserved tail of IgG memory–type B cell antigen receptors

A Double-Edged Kinase Lyn: A Positive and Negative Regulator for Antigen Receptor–mediated Signals

Email alerts

Update search

Synergistic cooperation and crosstalk between MYD88L265P and mutations that dysregulate CD79B and surface IgM

Consequences of the recurrent MYD88L265P somatic mutation for B cell tolerance

B cell survival, surface BCR and BAFFR expression, CD74 metabolism, and CD8− dendritic cells require the intramembrane endopeptidase SPPL2A

Human lymphoma mutations reveal CARD11 as the switch between self-antigen–induced B cell death or proliferation and autoantibody production

CD83 increases MHC II and CD86 on dendritic cells by opposing IL-10–driven MARCH1-mediated ubiquitination and degradation

Enhancement and suppression of signaling by the conserved tail of IgG memory–type B cell antigen receptors

A Double-Edged Kinase Lyn: A Positive and Negative Regulator for Antigen Receptor–mediated Signals

Email alerts

This Feature Is Available To Subscribers Only

Synergistic cooperation and crosstalk between MYD88^L265P and mutations that dysregulate CD79B and surface IgM

Consequences of the recurrent MYD88^L265P somatic mutation for B cell tolerance

B cell survival, surface BCR and BAFFR expression, CD74 metabolism, and CD8⁻ dendritic cells require the intramembrane endopeptidase SPPL2A