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Journal Articles

Alternative end-joining catalyzes class switch recombination in the absence of both Ku70 and DNA ligase 4

Cristian Boboila, Catherine Yan, Duane R. Wesemann, Mila Jankovic, Jing H. Wang, John Manis, Andre Nussenzweig, Michel Nussenzweig, Frederick W. Alt

J Exp Med (2013) 210 (3): 641.

https://doi.org/10.1084/jem.200924492103c

Published: 11 March 2013

View article titled, Alternative end-joining catalyzes class switch recombination in the absence of both Ku70 and DNA ligase 4

Journal Articles

Epigenetic tethering of AID to the donor switch region during immunoglobulin class switch recombination

Beena Patricia Jeevan-Raj, Isabelle Robert, Vincent Heyer, Adeline Page, Jing H. Wang, Florence Cammas, Frederick W. Alt, Régine Losson, Bernardo Reina-San-Martin

J Exp Med (2011) 208 (8): 1649–1660.

https://doi.org/10.1084/jem.20110118

Published: 11 July 2011

View article titled, Epigenetic tethering of AID to the donor switch region during immunoglobulin class switch recombination

Includes: Supplementary data

Journal Articles

Alternative end-joining catalyzes class switch recombination in the absence of both Ku70 and DNA ligase 4

Cristian Boboila, Catherine Yan, Duane R. Wesemann, Mila Jankovic, Jing H. Wang, John Manis, Andre Nussenzweig, Michel Nussenzweig, Frederick W. Alt

J Exp Med (2010) 207 (2): 417–427.

https://doi.org/10.1084/jem.20092449

Published: 08 February 2010

View article titled, Alternative end-joining catalyzes class switch recombination in the absence of both Ku70 and DNA ligase 4

Includes: Supplementary data

Journal Articles

Oncogenic transformation in the absence of Xrcc4 targets peripheral B cells that have undergone editing and switching

Jing H. Wang, Frederick W. Alt, Monica Gostissa, Abhishek Datta, Michael Murphy, Marat B. Alimzhanov, Kristen M. Coakley, Klaus Rajewsky, John P. Manis, Catherine T. Yan

J Exp Med (2008) 205 (13): 3079–3090.

https://doi.org/10.1084/jem.20082271

Published: 08 December 2008

View article titled, Oncogenic transformation in the absence of Xrcc4 targets peripheral B cells that have undergone editing and switching

Includes: Supplementary data