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Journal Articles

Tumor necrosis factor alpha is involved in mouse growth and lymphoid tissue development.

S de Kossodo, G E Grau, T Daneva, P Pointaire, L Fossati, C Ody, J Zapf, P F Piguet, R C Gaillard, P Vassalli

J Exp Med (1992) 176 (5): 1259–1264.

https://doi.org/10.1084/jem.176.5.1259

Published: 01 November 1992

View article titled, Tumor necrosis factor alpha is involved in mouse growth and lymphoid tissue development.

Journal Articles

Cytokines kill malaria parasites during infection crisis: extracellular complementary factors are essential.

T S Naotunne, N D Karunaweera, G Del Giudice, M U Kularatne, G E Grau, R Carter, K N Mendis

J Exp Med (1991) 173 (3): 523–529.

https://doi.org/10.1084/jem.173.3.523

Published: 01 March 1991

View article titled, Cytokines kill malaria parasites during infection crisis: extracellular complementary factors are essential.

Journal Articles

Tumor necrosis factor is a critical mediator in hapten induced irritant and contact hypersensitivity reactions.

P F Piguet, G E Grau, C Hauser, P Vassalli

J Exp Med (1991) 173 (3): 673–679.

https://doi.org/10.1084/jem.173.3.673

Published: 01 March 1991

View article titled, Tumor necrosis factor is a critical mediator in hapten induced irritant and contact hypersensitivity reactions.

Journal Articles

Interleukin 6 production in experimental cerebral malaria: modulation by anticytokine antibodies and possible role in hypergammaglobulinemia.

G E Grau, K Frei, P F Piguet, A Fontana, H Heremans, A Billiau, P Vassalli, P H Lambert

J Exp Med (1990) 172 (5): 1505–1508.

https://doi.org/10.1084/jem.172.5.1505

Published: 01 November 1990

View article titled, Interleukin 6 production in experimental cerebral malaria: modulation by anticytokine antibodies and possible role in hypergammaglobulinemia.

Journal Articles

Association between protective efficacy of anti-lipopolysaccharide (LPS) antibodies and suppression of LPS-induced tumor necrosis factor alpha and interleukin 6. Comparison of O side chain-specific antibodies with core LPS antibodies.

J D Baumgartner, D Heumann, J Gerain, P Weinbreck, G E Grau, M P Glauser

J Exp Med (1990) 171 (3): 889–896.

https://doi.org/10.1084/jem.171.3.889

Published: 01 March 1990

View article titled, Association between protective efficacy of anti-lipopolysaccharide (LPS) antibodies and suppression of LPS-induced tumor necrosis factor alpha and interleukin 6. Comparison of O side chain-specific antibodies with core LPS antibodies.

Journal Articles

Tumor necrosis factor/cachectin plays a key role in bleomycin-induced pneumopathy and fibrosis.

P F Piguet, M A Collart, G E Grau, Y Kapanci, P Vassalli

J Exp Med (1989) 170 (3): 655–663.

https://doi.org/10.1084/jem.170.3.655

Published: 01 September 1989

View article titled, Tumor necrosis factor/cachectin plays a key role in bleomycin-induced pneumopathy and fibrosis.

Journal Articles

Prevention of experimental cerebral malaria by anticytokine antibodies. Interleukin 3 and granulocyte macrophage colony-stimulating factor are intermediates in increased tumor necrosis factor production and macrophage accumulation.

G E Grau, V Kindler, P F Piguet, P H Lambert, P Vassalli

J Exp Med (1988) 168 (4): 1499–1504.

https://doi.org/10.1084/jem.168.4.1499

Published: 01 October 1988

View article titled, Prevention of experimental cerebral malaria by anticytokine antibodies. Interleukin 3 and granulocyte macrophage colony-stimulating factor are intermediates in increased tumor necrosis factor production and macrophage accumulation.

Journal Articles

Tumor necrosis factor/cachectin is an effector of skin and gut lesions of the acute phase of graft-vs.-host disease.

P F Piguet, G E Grau, B Allet, P Vassalli

J Exp Med (1987) 166 (5): 1280–1289.

https://doi.org/10.1084/jem.166.5.1280

Published: 01 November 1987

View article titled, Tumor necrosis factor/cachectin is an effector of skin and gut lesions of the acute phase of graft-vs.-host disease.

Journal Articles

Induction of acute thrombocytopenia and infection of megakaryocytes by Rauscher murine leukemia virus reflect the genetic susceptibility to leukemogenesis.

G E Grau, P Puthavathana, S Izui, P H Lambert

J Exp Med (1983) 157 (3): 1028–1039.

https://doi.org/10.1084/jem.157.3.1028

Published: 01 March 1983

View article titled, Induction of acute thrombocytopenia and infection of megakaryocytes by Rauscher murine leukemia virus reflect the genetic susceptibility to leukemogenesis.

NARROW

Tumor necrosis factor alpha is involved in mouse growth and lymphoid tissue development.

Cytokines kill malaria parasites during infection crisis: extracellular complementary factors are essential.

Tumor necrosis factor is a critical mediator in hapten induced irritant and contact hypersensitivity reactions.

Interleukin 6 production in experimental cerebral malaria: modulation by anticytokine antibodies and possible role in hypergammaglobulinemia.

Association between protective efficacy of anti-lipopolysaccharide (LPS) antibodies and suppression of LPS-induced tumor necrosis factor alpha and interleukin 6. Comparison of O side chain-specific antibodies with core LPS antibodies.

Tumor necrosis factor/cachectin plays a key role in bleomycin-induced pneumopathy and fibrosis.

Prevention of experimental cerebral malaria by anticytokine antibodies. Interleukin 3 and granulocyte macrophage colony-stimulating factor are intermediates in increased tumor necrosis factor production and macrophage accumulation.

Tumor necrosis factor/cachectin is an effector of skin and gut lesions of the acute phase of graft-vs.-host disease.

Induction of acute thrombocytopenia and infection of megakaryocytes by Rauscher murine leukemia virus reflect the genetic susceptibility to leukemogenesis.

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