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Journal Articles

Plastin 3 rescues cell surface translocation and activation of TrkB in spinal muscular atrophy

Luisa Hennlein, Hanaa Ghanawi, Florian Gerstner, Eduardo Palominos García, Ezgi Yildirim, Lena Saal-Bauernschubert, Mehri Moradi, Chunchu Deng, Teresa Klein, Silke Appenzeller, Markus Sauer, Michael Briese, Christian Simon, Michael Sendtner, Sibylle Jablonka

J Cell Biol (2023) 222 (3): e202204113.

https://doi.org/10.1083/jcb.202204113

Published: 06 January 2023

View article titled, Plastin 3 rescues cell surface translocation and activation of TrkB in spinal muscular atrophy

Includes: Supplementary data

Journal Articles

Differential roles of α-, β-, and γ-actin in axon growth and collateral branch formation in motoneurons

Mehri Moradi, Rajeeve Sivadasan, Lena Saal, Patrick Lüningschrör, Benjamin Dombert, Reena Jagdish Rathod, Daniela C. Dieterich, Robert Blum, Michael Sendtner

J Cell Biol (2017) 216 (3): 793–814.

https://doi.org/10.1083/jcb.201604117

Published: 28 February 2017

View article titled, Differential roles of α-, β-, and γ-actin in axon growth and collateral branch formation in motoneurons

Includes: Multimedia, Supplementary data

Journal Articles

Local axonal function of STAT3 rescues axon degeneration in the pmn model of motoneuron disease

Bhuvaneish Thangaraj Selvaraj, Nicolas Frank, Florian L.P. Bender, Esther Asan, Michael Sendtner

J Cell Biol (2012) 199 (3): 437–451.

https://doi.org/10.1083/jcb.201203109

Published: 29 October 2012

View article titled, Local axonal function of STAT3 rescues axon degeneration in the pmn model of motoneuron disease

Includes: Supplementary data

Journal Articles

Defective Ca²⁺ channel clustering in axon terminals disturbs excitability in motoneurons in spinal muscular atrophy

Sibylle Jablonka, Marcus Beck, Barbara Dorothea Lechner, Christine Mayer, Michael Sendtner

J Cell Biol (2007) 179 (1): 139–149.

https://doi.org/10.1083/jcb.200703187

Published: 08 October 2007

View article titled, Defective Ca2+ channel clustering in axon terminals disturbs excitability in motoneurons in spinal muscular atrophy

Includes: Supplementary data

Journal Articles

The p75NTR-interacting protein SC1 inhibits cell cycle progression by transcriptional repression of cyclin E

Alexandra Chittka, Juan Carlos Arevalo, Maria Rodriguez-Guzman, Pilar Pérez, Moses V. Chao, Michael Sendtner

J Cell Biol (2004) 164 (7): 985–996.

https://doi.org/10.1083/jcb.200301106

Published: 29 March 2004

View article titled, The p75NTR-interacting protein SC1 inhibits cell cycle progression by transcriptional repression of cyclin E

Journal Articles

Smn, the spinal muscular atrophy–determining gene product, modulates axon growth and localization of β-actin mRNA in growth cones of motoneurons

In Special Collection:

JCB65: RNA

Wilfried Rossoll, Sibylle Jablonka, Catia Andreassi, Ann-Kathrin Kröning, Kathrin Karle, Umrao R. Monani, Michael Sendtner

J Cell Biol (2003) 163 (4): 801–812.

https://doi.org/10.1083/jcb.200304128

Published: 17 November 2003

View article titled, Smn, the spinal muscular atrophy–determining gene product, modulates axon growth and localization of β-actin mRNA in growth cones of motoneurons

Includes: Supplementary data

Journal Articles

A transgene carrying an A2G missense mutation in the SMN gene modulates phenotypic severity in mice with severe (type I) spinal muscular atrophy

Umrao R. Monani, Matthew T. Pastore, Tatiana O. Gavrilina, Sibylle Jablonka, Thanh T. Le, Catia Andreassi, Jennifer M. DiCocco, Christian Lorson, Elliot J. Androphy, Michael Sendtner, Michael Podell, Arthur H.M. Burghes

J Cell Biol (2003) 160 (1): 41–52.

https://doi.org/10.1083/jcb.200208079

Published: 06 January 2003

View article titled, A transgene carrying an A2G missense mutation in the SMN gene modulates phenotypic severity in mice with severe (type I) spinal muscular atrophy

Includes: Supplementary data

Journal Articles

Missense mutation in the tubulin-specific chaperone E (Tbce) gene in the mouse mutant progressive motor neuronopathy, a model of human motoneuron disease

Heike Bömmel, Gang Xie, Wilfried Rossoll, Stefan Wiese, Sibylle Jablonka, Thomas Boehm, Michael Sendtner

J Cell Biol (2002) 159 (4): 563–569.

https://doi.org/10.1083/jcb.200208001

Published: 25 November 2002

View article titled, Missense mutation in the tubulin-specific chaperone E (Tbce) gene in the mouse mutant progressive motor neuronopathy, a model of human motoneuron disease

Includes: Supplementary data

Journal Articles

Conditional gene ablation of Stat3 reveals differential signaling requirements for survival of motoneurons during development and after nerve injury in the adult

Ulrich Schweizer, Jennifer Gunnersen, Christoph Karch, Stefan Wiese, Bettina Holtmann, Kiyoshi Takeda, Shizuo Akira, Michael Sendtner

J Cell Biol (2002) 156 (2): 287–298.

https://doi.org/10.1083/jcb.200107009

Published: 21 January 2002

View article titled, Conditional gene ablation of Stat3 reveals differential signaling requirements for survival of motoneurons during development and after nerve injury in the adult

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Plastin 3 rescues cell surface translocation and activation of TrkB in spinal muscular atrophy

Differential roles of α-, β-, and γ-actin in axon growth and collateral branch formation in motoneurons

Local axonal function of STAT3 rescues axon degeneration in the pmn model of motoneuron disease

Defective Ca²⁺ channel clustering in axon terminals disturbs excitability in motoneurons in spinal muscular atrophy

The p75NTR-interacting protein SC1 inhibits cell cycle progression by transcriptional repression of cyclin E

Smn, the spinal muscular atrophy–determining gene product, modulates axon growth and localization of β-actin mRNA in growth cones of motoneurons

A transgene carrying an A2G missense mutation in the SMN gene modulates phenotypic severity in mice with severe (type I) spinal muscular atrophy

Missense mutation in the tubulin-specific chaperone E (Tbce) gene in the mouse mutant progressive motor neuronopathy, a model of human motoneuron disease

Conditional gene ablation of Stat3 reveals differential signaling requirements for survival of motoneurons during development and after nerve injury in the adult

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