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Kim Newton
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Journal Articles

NLRP3 recruitment by NLRC4 during Salmonella infection

Yan Qu, Shahram Misaghi, Kim Newton, Allie Maltzman, Anita Izrael-Tomasevic, David Arnott, Vishva M. Dixit
Journal: Journal of Experimental Medicine
J Exp Med (2016) 213 (6): 877–885.
https://doi.org/10.1084/jem.20132234
Published: 02 May 2016
View article titled, NLRP3 recruitment by NLRC4 during Salmonella infection
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Journal Articles

JNK-mediated phosphorylation of DLK suppresses its ubiquitination to promote neuronal apoptosis

Sarah Huntwork-Rodriguez, Bei Wang, Trent Watkins, Arundhati Sengupta Ghosh, Christine D. Pozniak, Daisy Bustos, Kim Newton, Donald S. Kirkpatrick, Joseph W. Lewcock
Journal: Journal of Cell Biology
J Cell Biol (2013) 202 (5): 747–763.
https://doi.org/10.1083/jcb.201303066
Published: 26 August 2013
View article titled, JNK-mediated phosphorylation of DLK suppresses its ubiquitination to promote neuronal apoptosis
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Includes: Supplementary data
Journal Articles

Redundant roles for inflammasome receptors NLRP3 and NLRC4 in host defense against Salmonella

Petr Broz, Kim Newton, Mohamed Lamkanfi, Sanjeev Mariathasan, Vishva M. Dixit, Denise M. Monack
Journal: Journal of Experimental Medicine
J Exp Med (2010) 207 (8): 1745–1755.
https://doi.org/10.1084/jem.20100257
Published: 05 July 2010
View article titled, Redundant roles for inflammasome receptors NLRP3 and NLRC4 in host defense against Salmonella
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Includes: Supplementary data
Journal Articles

Ionizing Radiation and Chemotherapeutic Drugs Induce Apoptosis in Lymphocytes in the Absence of FAS or Fadd/Mort1 Signaling: Implications for Cancer Therapy

Kim Newton, Andreas Strasser
Journal: Journal of Experimental Medicine
J Exp Med (2000) 191 (1): 195–200.
https://doi.org/10.1084/jem.191.1.195
Published: 03 January 2000
View article titled, Ionizing Radiation and Chemotherapeutic Drugs Induce Apoptosis in Lymphocytes in the Absence of FAS or Fadd/Mort1 Signaling: Implications for Cancer Therapy
Open the PDF for Ionizing Radiation and Chemotherapeutic Drugs Induce Apoptosis in Lymphocytes in the Absence of FAS or Fadd/Mort1 Signaling: Implications for Cancer Therapy in another window