Figure 3.
Panel of CT angiogram images showing vascular involvement and stenotic lesions and PET scan revealing non-necrotic lympadenopathy in patient 3. (a, c, and d) CT angiography of abdominal arteries of patient 3 shows concentric wall thickening from the level of the aortic hiatus, the entire suprarenal and renal segments (arrows labelled as 1 to 4 in a to d), and the short infrarenal segment of the abdominal aorta for a length of 9.0–9.5 cm and maximum wall thickness of 3.2 mm. (b–d) Mild eccentric narrowing at the celiac artery origin of about 20–30% stenosis, severe short-segmental stenosis/near-total occlusion at the origin of the anterior right renal artery (c), and long segmental occlusion for a length of 14 mm of the posterior right renal artery (d). The rest of the infrarenal abdominal aorta, aortic bifurcation, and both iliac arteries appear normal in course, caliber, outline, and branching pattern. (e and f) Right kidney (measures 7.5 cm) shows mild diffuse shrinkage in volume with mild hypoperfusion of the right kidney with delayed excretion, whereas the left kidney (measures 11.1 cm) shows compensatory enlargement, and normal perfusion and normal excretion. (g) Positron emission tomography scan shows no evidence of abnormal FDG uptake at concentric wall thickening/stenosis involving abdominal aorta and its branches. Multiple FDG avid patchy consolidation changes are seen mainly in the subpleural and few in the peribronchial regions of the lower aspect of the right middle lobe, and all basal segments of the right lower lobe. FDG avid small to mildly enlarged non-necrotic nodes are seen at right paratracheal, pretracheal, precarinal, subcarinal, right hilar, and right inferior pulmonary ligament, and lower paraesophageal regions may be noted. Refer to the image caption for details. Panel a shows a CT angiogram of the abdominal arteries with concentric wall thickening. Panel b highlights the celiac artery origin with mild eccentric narrowing. Panels c and d provide additional views of the abdominal aorta with wall thickening. Panel e displays the right kidney with mild shrinkage and hypoperfusion, while Panel f shows the left kidney with compensatory enlargement and normal perfusion. Panel g presents a PET scan with no abnormal FDG uptake at the aortic stenosis but shows FDG avid consolidation changes in the lungs and enlarged lymph nodes in various regions.

Panel of CT angiogram images showing vascular involvement and stenotic lesions and PET scan revealing non-necrotic lympadenopathy in patient 3. (a, c, and d) CT angiography of abdominal arteries of patient 3 shows concentric wall thickening from the level of the aortic hiatus, the entire suprarenal and renal segments (arrows labelled as 1 to 4 in a to d), and the short infrarenal segment of the abdominal aorta for a length of 9.0–9.5 cm and maximum wall thickness of 3.2 mm. (b–d) Mild eccentric narrowing at the celiac artery origin of about 20–30% stenosis, severe short-segmental stenosis/near-total occlusion at the origin of the anterior right renal artery (c), and long segmental occlusion for a length of 14 mm of the posterior right renal artery (d). The rest of the infrarenal abdominal aorta, aortic bifurcation, and both iliac arteries appear normal in course, caliber, outline, and branching pattern. (e and f) Right kidney (measures 7.5 cm) shows mild diffuse shrinkage in volume with mild hypoperfusion of the right kidney with delayed excretion, whereas the left kidney (measures 11.1 cm) shows compensatory enlargement, and normal perfusion and normal excretion. (g) Positron emission tomography scan shows no evidence of abnormal FDG uptake at concentric wall thickening/stenosis involving abdominal aorta and its branches. Multiple FDG avid patchy consolidation changes are seen mainly in the subpleural and few in the peribronchial regions of the lower aspect of the right middle lobe, and all basal segments of the right lower lobe. FDG avid small to mildly enlarged non-necrotic nodes are seen at right paratracheal, pretracheal, precarinal, subcarinal, right hilar, and right inferior pulmonary ligament, and lower paraesophageal regions may be noted.

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