Figure 3.

Formation of ER–FA contacts is dependent on the binding of Rab18 and KIF5B to KNT1. (a) U2OS cells silenced with siRNA control or siRNA targeting Rab18 were cotransfected with ER-GFP and RFP-vinculin. The images represent maximum-intensity projections from Z-stacks. The insets show magnifications of the boxed regions. Scale bar: 10 µm; insets: 2 µm. (b) Quantification of the percentage of FAs in contact with the ER. The graph represents the mean ± SEM from three independent experiments (n > 30 cells). ***, P < 0.001. (c) U2OS cells were treated with either control siRNA or siRNA directed against KNT1 and transfected with either dsRed-ER alone or together with either GFP KNT1 FL, TM, or Δ201–445. Cells were fixed and stained with anti-vinculin antibody. The images represent maximum-intensity projections from Z-stacks. The insets show magnification of boxed regions to illustrate contact points between ER and FAs. Scale bars: 10 µm; insets: 2 µm. (d) Quantification of the percentage of FAs in contact with the ER. The graph represents the mean ± SEM from three independent experiments (n > 30 cells). *, P < 0.05; ***, P ≤ 0.001; n.s., not significant.

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