Figure S5.
Genes related to microbial sensing, keratin production, opsonization, complement activation, and periodontitis induced upon oral TRMreactivation are blunted upon CD103+TRMdepletion. (A–G) Highlighted are genes for TLRs (A), keratin production (B), antibody-dependent cell-mediated cytotoxicity (C), complement (D), olfaction (E), steriocillin (F), and periodontitis (G). As in Fig. 8, gray circles represent gp33-treated animals, red circles represent SIIN-treated animals, black circles represent SIIN-103–treated animals. Differences between gp33 vs. SIIN and SIIN vs. SIIN-103 are all statistically significant (*, P < 0.05; as determined by a Student’s t test between the relevant comparisons) unless otherwise indicated, in which case P values are shown. Dots represent individual mice per group. N = 3 (gp33) or 4 (SIIN, SIIN-103) mice per treatment group.

Genes related to microbial sensing, keratin production, opsonization, complement activation, and periodontitis induced upon oral T RM reactivation are blunted upon CD103 + T RM depletion. (A–G) Highlighted are genes for TLRs (A), keratin production (B), antibody-dependent cell-mediated cytotoxicity (C), complement (D), olfaction (E), steriocillin (F), and periodontitis (G). As in Fig. 8, gray circles represent gp33-treated animals, red circles represent SIIN-treated animals, black circles represent SIIN-103–treated animals. Differences between gp33 vs. SIIN and SIIN vs. SIIN-103 are all statistically significant (*, P < 0.05; as determined by a Student’s t test between the relevant comparisons) unless otherwise indicated, in which case P values are shown. Dots represent individual mice per group. N = 3 (gp33) or 4 (SIIN, SIIN-103) mice per treatment group.

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