Figure S2.
Sequencing results of capLECs, colLECs, and BECs derived from control and CHS-inflamed skin.(A) Gating strategy used for FACS sorting of singlet, 7AAD− capLECs (CD45−CD31+podo+LYVE-1+), colLECs (CD45−CD31+podo+LYVE-1−), and BECs (CD45−CD31+podo− LYVE-1−). (B and C) Euler diagrams showing number of differentially expressed genes (0.5-fold; P value, 0.05) in BECs versus colLECs/BECs versus capLECs/colLECs versus capLECs in CTR skin (B) and in INF skin (C). (D) Sample clustering of capLECs, colLECs, and BECs from INF and CTR mice showing the top 100 most variable genes. (E) Heatmap showing lymphatic, blood, and pan-endothelial genes in capLECs, colLECs, and BECs from INF and CTR mice (n = 5 experimental replicates). FSC-A, forward scatter area; FSC-H, forward scatter height; podo, podoplanin; SSC-A, side scatter area.

Sequencing results of capLECs, colLECs, and BECs derived from control and CHS-inflamed skin. (A) Gating strategy used for FACS sorting of singlet, 7AAD− capLECs (CD45−CD31+podo+LYVE-1+), colLECs (CD45−CD31+podo+LYVE-1−), and BECs (CD45−CD31+podo− LYVE-1−). (B and C) Euler diagrams showing number of differentially expressed genes (0.5-fold; P value, 0.05) in BECs versus colLECs/BECs versus capLECs/colLECs versus capLECs in CTR skin (B) and in INF skin (C). (D) Sample clustering of capLECs, colLECs, and BECs from INF and CTR mice showing the top 100 most variable genes. (E) Heatmap showing lymphatic, blood, and pan-endothelial genes in capLECs, colLECs, and BECs from INF and CTR mice (n = 5 experimental replicates). FSC-A, forward scatter area; FSC-H, forward scatter height; podo, podoplanin; SSC-A, side scatter area.

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