Figure 8.
Moesin deficiency translates in bone defects. (A) Representative x-ray images of the whole skeleton of WT and Msn−/− mice. Scale bar, 5 cm. (B) Representative microcomputed tomography images of trabecular section of distal femurs from WT mice and Msn−/− mice. Scale bar, 500 µm. (C) Histograms indicate means ± SD of trabecular bone volume per total volume (BV/TV), trabecular thickness (Tb.Th), number (Tb.N), and separation (Tb.Sp), analyzed by microcomputed tomography. (D) Histograms indicate means ± SD of cortical bone parameters, analyzed by microcomputed tomography. (B–D) Animal groups were composed of 6 mice of each genotype. In C, each mouse is represented by one color. 12 femora were analyzed in total for Msn−/− mice and 10 femora for the WT mice group. n.s., not significant.

Moesin deficiency translates in bone defects. (A) Representative x-ray images of the whole skeleton of WT and Msn−/− mice. Scale bar, 5 cm. (B) Representative microcomputed tomography images of trabecular section of distal femurs from WT mice and Msn−/− mice. Scale bar, 500 µm. (C) Histograms indicate means ± SD of trabecular bone volume per total volume (BV/TV), trabecular thickness (Tb.Th), number (Tb.N), and separation (Tb.Sp), analyzed by microcomputed tomography. (D) Histograms indicate means ± SD of cortical bone parameters, analyzed by microcomputed tomography. (B–D) Animal groups were composed of 6 mice of each genotype. In C, each mouse is represented by one color. 12 femora were analyzed in total for Msn−/− mice and 10 femora for the WT mice group. n.s., not significant.

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