LCMV Cl13 infection neither increases intestinal permeability to FD40 nor causes epithelial layer disruption. Related to Fig. 1. C57BL/6 mice were infected with LCMV ARM or Cl13 or left uninfected (Un) and analyzed at indicated time points p.i. (A) In vivo intestinal permeability to FD40 was measured in ARM- and Cl13-infected mice and normalized to levels in uninfected mice. (B and C) Hematoxylin and eosin staining of small (B) and large (C) intestinal sections. All scale bars correspond to 72 µm. (A) Averages ± SEM are shown. Data in A are pooled from two experimental repeats where statistical significance was achieved in one out of two experiments or upon pooling. Data in B and C are representative of two independent experimental repeats. *, P < 0.05; Kruskal–Wallis with Dunn’s multiple comparisons correction (A).