Table 1.

Comparison of genetic and/or pharmacologic CTLA-4 loss in humans and mice

Disease featureHumanMouse
CTLA4−/−CTLA4+/−Ipilimumab-treatedCtla4−/−Ctla4+/−
Genetic variant Homozygous Ser172Pro (hypomorphic, n = 1) Heterozygous loss-of-function Not applicable Homozygous loss-of-function Heterozygous loss-of-function 
Outcome Alive at 9 years old on abatacept, immunoglobulin replacement, and prophylactic antibiotics Variable disease penetrance (∼70% affected). Most respond to sirolimus or abatacept and replacement immunoglobulin if needed. Hematopoietic stem cell transplantation is an option for severe cases Immune-related adverse effects appear within a few weeks to months after treatment. Most resolve upon delaying dose or with immunosuppressants Death by ∼3–7 wk of age Not reported (healthy) 
Lymphoproliferation Hepatosplenomegaly Lymphadenopathy, hepatosplenomegaly None Hepatosplenomegaly None (healthy) 
Lymphocytic infiltration Enteropathy (celiac disease) Enteropathy, inflammatory bowel disease, granulomatous-lymphocytic interstitial lung disease, lymphocytic infiltrations in the brain, bone marrow, kidney, atopic dermatitis, and arthritis > pancreas Dermatitis, enterocolitis, hepatitis, and pneumonitis Multiorgan mononuclear infiltrates in heart (with cardiomegaly), pancreas, lungs, salivary glands, liver, bone marrow > synovium, and blood vessels None (healthy) 
​ ​ ​ Increased activated T cells in spleen, lymph nodes, thymus, and increased activated B cells in spleen and lymph nodes ​ 
Autoimmunity Immune thrombocytopenic purpura Immune thrombocytopenic purpura, autoimmune hemolytic anemia, autoimmune neutropenia, thyroiditis/hypothyroidism, type 1 diabetes mellitus, psoriasis, vitiligo, and alopecia Hypophysitis with pituitary autoantibodies, thyroiditis/hypothyroidism, and rarely autoimmune cytopenias Anti-insulin, gastric parietal, Ro52, and dsDNA autoantibodies (when Ctla4 is conditionally deleted in Treg) None (healthy) 
​ ​ ​ Worsened collagen-induced arthritis disease, but delayed or ameliorated experimental allergic encephalitis (when Ctla4 is conditionally deleted in Treg) ​ 
Infection susceptibility Mild recurrent upper respiratory tract infections Recurrent sinopulmonary (±bronchiectasis) and otitis media infections Infrequent None reported None (healthy) 
​ Elevated EBV DNA copy number (while on methylprednisolone, azathioprine) ​ ​ ​ ​ 
Other ​ Lymphoma, gastric cancer ​ ​ ​ 
Laboratory abnormalities Increased circulating Tfh, effector memory and exhausted T cells Normal or low numbers of total lymphocytes, T cells, CD4+ T cells, CD8+ T cells, Treg, B cells, and NK cells Not investigated/reported Spontaneous and TCR-stimulated proliferation and cytokine production Increased germinal centers after immunization 
Increased IFN-γ and IL-10 expression in T cells Decreased numbers of Treg having impaired suppressor function, naïve T cells, and memory B cells ​ Increased IFN-γ, IL-4, and GM-CSF upon TCR stimulation ​ 
Increased activated and atypical memory B cells Increased numbers of activated memory T cells and circulating TFH cells Increased Treg, Tfh, and Tfr (but Tfr are functionally impaired) 
Hypogammaglobulinemia, poor vaccine titers Hypogammaglobulinemia, poor vaccine titers Hypergammaglobulinemia 
References This article in JHI (1) (2, 3, 4, 5) (2) (6, 7, 8) (7, 9) 

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