Table 1.

Summary of microglia and TREM2 effects on tau-linked pathologies in animal models

ConditionModelAgeTau propagation and tau pathologyNeurodegenerationGlial reactivityReference
Microglial depletion Aβ models 5xFAD × P301S + injection of recombinant tau seeds 7 mo ↓ AT8 area (CX, HC) ↓ Neurodegeneration (CX, HC) Remaining microglia exhibit DAM phenotype No difference Lodder et al. (2021)  
AppNL-G-F+ AAV-P301L-tau injection 6 mo ↓ AT8 propagation
↑ AT8 plaque area 
↓ MGnD+ total and plaque area (Clec7a) ↑ SP N
↑ SP areas
↑ SP size 
Clayton et al. (2021)  
5xFAD + injection of AD brain–derived tau 9.5 mo ↑ AT8 seeding and propagation
↑ AT8 area (CX, HC) 
No difference in microglial reactivity (CD68)
No difference in astrocyte clustering around plaques
↑ GFAP RNA expression 
No difference Gratuze et al. (2021)  
5xFAD + injection of AD brain–derived tau 6 mo ↓ AT8 signal (CX) ↓ SP load
↓ SP size 
Delizannis et al. (2021)  
Tau models P301S 4 mo ↓ AT8
↓ globular tau oligomers
↓ NFTs
No difference in AT8 or T-tau (ELISA) 
↓ Neurodegeneration ↓ Expression of proinflammatory cytokines Mancuso et al. (2019)  
P301S × APOE4 KI or apoE KO 9 mo ↓ AT8 area (HC)
↓ HJ14.5 (pSer396) levels (CX) 
↓ Neurodegeneration (entorhinal/piriform CX, HC) Remaining microglia exhibit reactive state (CD68) Shi et al. (2019)  
C57BL/6 + injection of AAV-GFP/tau 4.5 mo ↓ tau propagation (?)
↓ AT8+ cells (EC and DG) 
Asai et al. (2015)  
Tg4510 15 mo No differences No difference Microglia exhibit DAM phenotype (e.g., apoE, CD68)
No change in astrocyte reactivity (GFAP) 
Bennett et al. (2018)  
TREM2 depletion or R47H expression Aβ models APPPS1-21 × TREM2 KO Or TREM2 R47H + injection of AD brain–derived tau 8.5 mo ↑ AT8 seeding and propagation
↑ AT8 area (CX)
↑ AT8 signal near plaques 
↓ Microglial reactivity (Iba1) ↑ Aβ42 in and around SP (CX) Leyns et al. (2019)  
5xFAD + injection of AD brain–derived tau 9 mo ↑ AT8 seeding and propagation
↑ AT8 area (CX, HC) 
↓ Microglial reactivity (Iba1)
↓ Astrocytic reactivity (GFAP) 
↑ SP coverage (CX, HC) Gratuze et al. (2021)  
Tau models P301S + TREM2 shRNA injection 7 mo ↑ AT8, AT100, and AT180 ↑ Neurodegeneration (CX, HC) ↑ Expression of proinflammatory cytokines Jiang et al. (2015)  
htau × TREM2 KO 6 mo ↑ AT8 (CX, HC)
↑ AT180, PHF-1 (CX) 
Bemiller et al. (2017)  
P301S × TREM2 KO or TREM2 haploinsufficient mice 8–9 mo ↑ AT8 area (CX of haploinsufficient mice)
↑ MC1 area (HC of haploinsufficient mice)
No difference in TREM2 KO mice 
↓ Neurodegeneration (ventricle area) ↑ Microglial reactivity (based on morphology in haploinsufficient mice)
TREM2 KO microglial morphology similar to WT 
Sayed et al. (2018)  
THY-Tau22 × TREM2 KO 12 mo ↑ AT8 (HC)
↑ N° of AT100+ and Gallyas+ neurons 
↓ Neurodegeneration (surface CA1)
No difference in pyramidal nucleus layer thickness 
↓ Microglial reactivity (e.g., CD68, IL-1β)
No difference in astrocyte reactivity (GFAP) 
Vautheny et al. (2021)  
AAV-P301L tau injection 5 mo ↑ tau spreading ↑ Microglial reactivity (e.g., CD68, Clec7a, CD9 in DG)
↑ Microglial reactivity in MEC (CD9) 
Zhu et al. (2022)  
P301S x R47H 9 mo ↓ AT8 and PG5 area (HC)
↓ AT180 area (piriform CX) 
↓ Neurodegeneration (HC, entorhinal, piriform CX, pyramidal and DG layer thickness, ventricle volume) ↓ Microglial reactivity (Iba1, CD68)
↓ DAM gene expression
↓ Proinflammatory cytokines 
Gratuze et al. (2020)  
TREM2 KO + injection of AD brain–derived tau 9 mo ↓ AT8 area
No difference in HC 
↓ Microglial reactivity (Iba1)
↓ Neuroinflammatory response
No change in astrocyte reactivity (GFAP) 
Lee-Gosselin et al. (2023)  
P301S × TREM2 KO 9 mo No difference ↓ Neurodegeneration (entorhinal, piriform CX, ventricle volume) ↓ Microglial reactivity (Iba1)
↓ Inflammatory markers
Reduced astrocytic reactivity (GFAP) 
Leyns et al. (2017)  
TREM2R47H/TYROBP (Drosophila melanogaster7 d No difference No difference ↓ Inflammatory response Sekiya et al. (2018)  
TREM2 overexpression P301S + lentiviral-mediated TREM2 overexpression 7 mo ↓ AT8 and AT180 (CX, HC) ↓ Neurodegeneration (CX, HC) ↓ Proinflammatory cytokines
↑ Anti-inflammatory markers 
Jiang et al. (2016)  
ConditionModelAgeTau propagation and tau pathologyNeurodegenerationGlial reactivityReference
Microglial depletion Aβ models 5xFAD × P301S + injection of recombinant tau seeds 7 mo ↓ AT8 area (CX, HC) ↓ Neurodegeneration (CX, HC) Remaining microglia exhibit DAM phenotype No difference Lodder et al. (2021)  
AppNL-G-F+ AAV-P301L-tau injection 6 mo ↓ AT8 propagation
↑ AT8 plaque area 
↓ MGnD+ total and plaque area (Clec7a) ↑ SP N
↑ SP areas
↑ SP size 
Clayton et al. (2021)  
5xFAD + injection of AD brain–derived tau 9.5 mo ↑ AT8 seeding and propagation
↑ AT8 area (CX, HC) 
No difference in microglial reactivity (CD68)
No difference in astrocyte clustering around plaques
↑ GFAP RNA expression 
No difference Gratuze et al. (2021)  
5xFAD + injection of AD brain–derived tau 6 mo ↓ AT8 signal (CX) ↓ SP load
↓ SP size 
Delizannis et al. (2021)  
Tau models P301S 4 mo ↓ AT8
↓ globular tau oligomers
↓ NFTs
No difference in AT8 or T-tau (ELISA) 
↓ Neurodegeneration ↓ Expression of proinflammatory cytokines Mancuso et al. (2019)  
P301S × APOE4 KI or apoE KO 9 mo ↓ AT8 area (HC)
↓ HJ14.5 (pSer396) levels (CX) 
↓ Neurodegeneration (entorhinal/piriform CX, HC) Remaining microglia exhibit reactive state (CD68) Shi et al. (2019)  
C57BL/6 + injection of AAV-GFP/tau 4.5 mo ↓ tau propagation (?)
↓ AT8+ cells (EC and DG) 
Asai et al. (2015)  
Tg4510 15 mo No differences No difference Microglia exhibit DAM phenotype (e.g., apoE, CD68)
No change in astrocyte reactivity (GFAP) 
Bennett et al. (2018)  
TREM2 depletion or R47H expression Aβ models APPPS1-21 × TREM2 KO Or TREM2 R47H + injection of AD brain–derived tau 8.5 mo ↑ AT8 seeding and propagation
↑ AT8 area (CX)
↑ AT8 signal near plaques 
↓ Microglial reactivity (Iba1) ↑ Aβ42 in and around SP (CX) Leyns et al. (2019)  
5xFAD + injection of AD brain–derived tau 9 mo ↑ AT8 seeding and propagation
↑ AT8 area (CX, HC) 
↓ Microglial reactivity (Iba1)
↓ Astrocytic reactivity (GFAP) 
↑ SP coverage (CX, HC) Gratuze et al. (2021)  
Tau models P301S + TREM2 shRNA injection 7 mo ↑ AT8, AT100, and AT180 ↑ Neurodegeneration (CX, HC) ↑ Expression of proinflammatory cytokines Jiang et al. (2015)  
htau × TREM2 KO 6 mo ↑ AT8 (CX, HC)
↑ AT180, PHF-1 (CX) 
Bemiller et al. (2017)  
P301S × TREM2 KO or TREM2 haploinsufficient mice 8–9 mo ↑ AT8 area (CX of haploinsufficient mice)
↑ MC1 area (HC of haploinsufficient mice)
No difference in TREM2 KO mice 
↓ Neurodegeneration (ventricle area) ↑ Microglial reactivity (based on morphology in haploinsufficient mice)
TREM2 KO microglial morphology similar to WT 
Sayed et al. (2018)  
THY-Tau22 × TREM2 KO 12 mo ↑ AT8 (HC)
↑ N° of AT100+ and Gallyas+ neurons 
↓ Neurodegeneration (surface CA1)
No difference in pyramidal nucleus layer thickness 
↓ Microglial reactivity (e.g., CD68, IL-1β)
No difference in astrocyte reactivity (GFAP) 
Vautheny et al. (2021)  
AAV-P301L tau injection 5 mo ↑ tau spreading ↑ Microglial reactivity (e.g., CD68, Clec7a, CD9 in DG)
↑ Microglial reactivity in MEC (CD9) 
Zhu et al. (2022)  
P301S x R47H 9 mo ↓ AT8 and PG5 area (HC)
↓ AT180 area (piriform CX) 
↓ Neurodegeneration (HC, entorhinal, piriform CX, pyramidal and DG layer thickness, ventricle volume) ↓ Microglial reactivity (Iba1, CD68)
↓ DAM gene expression
↓ Proinflammatory cytokines 
Gratuze et al. (2020)  
TREM2 KO + injection of AD brain–derived tau 9 mo ↓ AT8 area
No difference in HC 
↓ Microglial reactivity (Iba1)
↓ Neuroinflammatory response
No change in astrocyte reactivity (GFAP) 
Lee-Gosselin et al. (2023)  
P301S × TREM2 KO 9 mo No difference ↓ Neurodegeneration (entorhinal, piriform CX, ventricle volume) ↓ Microglial reactivity (Iba1)
↓ Inflammatory markers
Reduced astrocytic reactivity (GFAP) 
Leyns et al. (2017)  
TREM2R47H/TYROBP (Drosophila melanogaster7 d No difference No difference ↓ Inflammatory response Sekiya et al. (2018)  
TREM2 overexpression P301S + lentiviral-mediated TREM2 overexpression 7 mo ↓ AT8 and AT180 (CX, HC) ↓ Neurodegeneration (CX, HC) ↓ Proinflammatory cytokines
↑ Anti-inflammatory markers 
Jiang et al. (2016)  

CX: cortex; HC: hippocampus; GFAP: Glial Fibrillary Acidic Protein; SP: senile plaque; EC: entorhinal cortex; DG: dentate gyrus.

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