Table 1.

Patients’ demographics and summary of clinical and immunological phenotype

SubjectCVID phenotypeSexRace/ethnicityAge at diagnosisAge at enrollmentTreatmentActive NIC at time of sample collectionHistory of immune suppression used# years from immune suppression use to fecal samplingBaseline IgABaseline IgMTotal B cellsCD27+ CD21lowCD3+CD4+CD8+
INF White 28 30 IVIG N/A N/A N/A <15 <5 6% (430) 9.5% (41) 75% (1,697) 31.9% (722) 40.3% (912) 
INF White 50 64 IVIG N/A Mesalamine 2 mo <5 <5 95% (1,949) 27.9% (572) 74% (1,518) 
INFa White 43 50 IVIG N/A Rituximab + hyper CVD 20 years <10 11 7.2% (98) 2.1% (1) 70% (1,084) 32.7% (445) 40.1 (545) 
15 INF White 52 57 SCIG N/A N/A N/A <5 8.5 18.2 (367) 21.5% (79) 77% (1,461) 44.3% (841) 31.7 (602) 
19 INF White  57 HyQvia N/A N/A N/A N/A N/A N/A N/A N/A N/A N/A 
NIC White 41 70 IVIG Sjogren syndrome, Barrett's esophagus, B12 deficiency, and chronic diarrhea N/A N/A <6 11.8 9.9% (198) 3.9% (8) 63% (1,106) 29% (509) 32% (562) 
NIC White/Hispanic 32 42 HyQvia CVID-entropathyc Budesonide On and off 3 years <5 <5 25.4% (283) 1% (4) 69% (770) 42% (469) 25.8% (288) 
11 NIC White 33 52 SCIG GLILD, liver NRH, CVID entropathy, and splenomegaly Ustekinumab and oral IgG 3 mo <6 7.3 15% (113) 3.1% (4) 73 (471) 40% (258) 27% (174) 
12 NICb White/Hispanic 34 38 IVIG Alopecia, vitiligo, premature ovarian failure, and cytopenia None N/A 76 76.3 N/A 80 47 31 
18 NIC White 59 64 IVIG GLILD, NRH, and thrombocytopenia Rituximab 2 years <9 339 21% (254) N/A 70% (892) 53% (627) 14% (168) 
20 NIC White  21 HyQvia Cerebral vasculitis and granulomatous encephalitis N/A N/A N/A N/A N/A N/A N/A N/A N/A 
SubjectCVID phenotypeSexRace/ethnicityAge at diagnosisAge at enrollmentTreatmentActive NIC at time of sample collectionHistory of immune suppression used# years from immune suppression use to fecal samplingBaseline IgABaseline IgMTotal B cellsCD27+ CD21lowCD3+CD4+CD8+
INF White 28 30 IVIG N/A N/A N/A <15 <5 6% (430) 9.5% (41) 75% (1,697) 31.9% (722) 40.3% (912) 
INF White 50 64 IVIG N/A Mesalamine 2 mo <5 <5 95% (1,949) 27.9% (572) 74% (1,518) 
INFa White 43 50 IVIG N/A Rituximab + hyper CVD 20 years <10 11 7.2% (98) 2.1% (1) 70% (1,084) 32.7% (445) 40.1 (545) 
15 INF White 52 57 SCIG N/A N/A N/A <5 8.5 18.2 (367) 21.5% (79) 77% (1,461) 44.3% (841) 31.7 (602) 
19 INF White  57 HyQvia N/A N/A N/A N/A N/A N/A N/A N/A N/A N/A 
NIC White 41 70 IVIG Sjogren syndrome, Barrett's esophagus, B12 deficiency, and chronic diarrhea N/A N/A <6 11.8 9.9% (198) 3.9% (8) 63% (1,106) 29% (509) 32% (562) 
NIC White/Hispanic 32 42 HyQvia CVID-entropathyc Budesonide On and off 3 years <5 <5 25.4% (283) 1% (4) 69% (770) 42% (469) 25.8% (288) 
11 NIC White 33 52 SCIG GLILD, liver NRH, CVID entropathy, and splenomegaly Ustekinumab and oral IgG 3 mo <6 7.3 15% (113) 3.1% (4) 73 (471) 40% (258) 27% (174) 
12 NICb White/Hispanic 34 38 IVIG Alopecia, vitiligo, premature ovarian failure, and cytopenia None N/A 76 76.3 N/A 80 47 31 
18 NIC White 59 64 IVIG GLILD, NRH, and thrombocytopenia Rituximab 2 years <9 339 21% (254) N/A 70% (892) 53% (627) 14% (168) 
20 NIC White  21 HyQvia Cerebral vasculitis and granulomatous encephalitis N/A N/A N/A N/A N/A N/A N/A N/A N/A 

GLILD: granulomatous lymphocytic interstitial lung disease. NRH: nodular regenerative hyperplasia. Hyper CVAD includes cyclophosphamide, vincristine, Adriamycin (doxorubicin), dexamethasone, methotrexate, and cytarabine. N/A: Data were not available for those patients.

a

Patient developed EBV-related lymphoma 20 years ago (currently in remission), treated with rituximab, never recovered Igs, has 0 switched memory B cells, undetectable IgA, and IgM.

b

Patient presented with CVID phenotype at age 34, later found to have RAG1 mutation.

c

CVID enteropathy includes inflammatory bowel disease like enteritis.

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