TABLE I

Parameters Used to Simulate Transient cAMP Signals Near the Plasma Membrane of HEK-293 Cells

Parameter
Parameter definition
Value
Initial condition
Reference
tot] Total β2AR concentration 1 μM  ** 
[β] Inactive receptor concentration  0.2 μM  
[β*] Activated receptor concentration  0.8 μM  
desense] Desensitized receptor concentration  0 μM  
int] Internalized receptor concentration  0 μM  
kGRK Rate constant of receptor phosphorylation 0.005 s−1  Tran et al., 2004  
krpp Rate constant of receptor dephosphorylation 0.0005 s−1  Iyer et al., 2006; Tran et al., 2007a  
kint Rate constant of receptor internalization 0.01 s−1   
krec Rate constant of receptor recycling 0.005 s−1   
kact Rate constant of Gs activation of β2AR 15 s−1  Frace et al., 1993  
kh Rate constant of GTP hydrolysis 0.8 s−1  Frace et al., 1993  
KD Dissociation constant of isoproterenol 250 nM  Whaley et al., 1994  
Gstot Total concentration of Gs 4 μM  ** with initial estimate from Post et al., 1995  
Kc Equilibrium constant between activated receptor and G protein 15 μM   
KGsAC Equilibrium constant between activated G protein and AC 315 μM   
ACsyn cAMP synthesis rate 10 μM·s−1  ** 
AC0 Basal cAMP synthesis rate 0.005 μM·s−1  ** 
[NcAMP concentration  0.5 μM Beavo et al., 1974  
Vmax-PDE Maximal hydrolysis rate of unphosphorylated PDE 0.15 μM⋅s−1  Reeves et al., 1987; Rich et al., 2001a, 2007  
Vmax-PDEp Maximal hydrolysis rate of phosphorylated PDE 2.5⋅ Vmax-PDE  Rich et al., 2007  
Km Michaelis constant for PDE 1 μM  Houslay et al., 1998  
KI Inhibition constant of rolipram 0.1 μM  Houslay et al., 1998; Richter and Conti, 2004  
K1/2 cAMP concentration that half maximally activates CNG channels 1.1 μM  Rich et al., 2001a,b  
N Hill coefficient of cAMP binding to CNG channels 2.1  Dhallan et al., 1990; Rich et al., 2001b  
kPKA Rate constant of PKA-mediated phosphorylation 0.015 μM−1·s−1  ** 
kpp Rate constant of PDE dephosphorylation 0.005 s−1  ** 
[PKA] PKA concentration 1 μM  Beavo et al., 1974; Hofmann et al., 1977; Rich and Karpen, 2002  
kfa Rate constant of cAMP binding to site a of the R subunit 5 μM−1·s−1  Doskeland and Ogreid, 1984; Rich and Karpen, 2002  
kfb Rate constant of cAMP binding to site b 0.4 μM−1·s−1  Doskeland and Ogreid, 1984; Rich and Karpen, 2002  
kra Rate constant of cAMP dissociation from site a 1 s−1  Doskeland and Ogreid, 1984; Houge et al., 1990; Rich and Karpen, 2002  
krb Rate constant of cAMP dissociation from site b 0.2 s−1  Doskeland and Ogreid, 1984; Houge et al., 1990; Rich and Karpen, 2002  
kPKAact Rate constant of R and C subunit dissociation 70 s−1  Smith et al., 1981; Houge et al., 1990; Huang and Taylor, 1998; Rich and Karpen, 2002  
kPKAdeact Rate constant of R and C subunit association 0.75 μM−2·s−1  Smith et al., 1981; Houge et al., 1990; Huang and Taylor, 1998; Rich and Karpen, 2002  
KI-PKI Inhibition constant of PKI 2.3 nM  Cheng et al., 1986  
Parameter
Parameter definition
Value
Initial condition
Reference
tot] Total β2AR concentration 1 μM  ** 
[β] Inactive receptor concentration  0.2 μM  
[β*] Activated receptor concentration  0.8 μM  
desense] Desensitized receptor concentration  0 μM  
int] Internalized receptor concentration  0 μM  
kGRK Rate constant of receptor phosphorylation 0.005 s−1  Tran et al., 2004  
krpp Rate constant of receptor dephosphorylation 0.0005 s−1  Iyer et al., 2006; Tran et al., 2007a  
kint Rate constant of receptor internalization 0.01 s−1   
krec Rate constant of receptor recycling 0.005 s−1   
kact Rate constant of Gs activation of β2AR 15 s−1  Frace et al., 1993  
kh Rate constant of GTP hydrolysis 0.8 s−1  Frace et al., 1993  
KD Dissociation constant of isoproterenol 250 nM  Whaley et al., 1994  
Gstot Total concentration of Gs 4 μM  ** with initial estimate from Post et al., 1995  
Kc Equilibrium constant between activated receptor and G protein 15 μM   
KGsAC Equilibrium constant between activated G protein and AC 315 μM   
ACsyn cAMP synthesis rate 10 μM·s−1  ** 
AC0 Basal cAMP synthesis rate 0.005 μM·s−1  ** 
[NcAMP concentration  0.5 μM Beavo et al., 1974  
Vmax-PDE Maximal hydrolysis rate of unphosphorylated PDE 0.15 μM⋅s−1  Reeves et al., 1987; Rich et al., 2001a, 2007  
Vmax-PDEp Maximal hydrolysis rate of phosphorylated PDE 2.5⋅ Vmax-PDE  Rich et al., 2007  
Km Michaelis constant for PDE 1 μM  Houslay et al., 1998  
KI Inhibition constant of rolipram 0.1 μM  Houslay et al., 1998; Richter and Conti, 2004  
K1/2 cAMP concentration that half maximally activates CNG channels 1.1 μM  Rich et al., 2001a,b  
N Hill coefficient of cAMP binding to CNG channels 2.1  Dhallan et al., 1990; Rich et al., 2001b  
kPKA Rate constant of PKA-mediated phosphorylation 0.015 μM−1·s−1  ** 
kpp Rate constant of PDE dephosphorylation 0.005 s−1  ** 
[PKA] PKA concentration 1 μM  Beavo et al., 1974; Hofmann et al., 1977; Rich and Karpen, 2002  
kfa Rate constant of cAMP binding to site a of the R subunit 5 μM−1·s−1  Doskeland and Ogreid, 1984; Rich and Karpen, 2002  
kfb Rate constant of cAMP binding to site b 0.4 μM−1·s−1  Doskeland and Ogreid, 1984; Rich and Karpen, 2002  
kra Rate constant of cAMP dissociation from site a 1 s−1  Doskeland and Ogreid, 1984; Houge et al., 1990; Rich and Karpen, 2002  
krb Rate constant of cAMP dissociation from site b 0.2 s−1  Doskeland and Ogreid, 1984; Houge et al., 1990; Rich and Karpen, 2002  
kPKAact Rate constant of R and C subunit dissociation 70 s−1  Smith et al., 1981; Houge et al., 1990; Huang and Taylor, 1998; Rich and Karpen, 2002  
kPKAdeact Rate constant of R and C subunit association 0.75 μM−2·s−1  Smith et al., 1981; Houge et al., 1990; Huang and Taylor, 1998; Rich and Karpen, 2002  
KI-PKI Inhibition constant of PKI 2.3 nM  Cheng et al., 1986  

Initial conditions were estimated based on steady-state parameter values in the presence of basal adenylyl cyclase activity. ** denotes values that were fit to the time course of cAMP signals as described in Rich et al. (2007). Concentrations of enzymes should be considered approximate, whether estimated or experimentally derived, because, with the exceptions of data provided from our work, experimentally derived data were obtained from other cellular systems, either overexpression systems or other cell types. In addition, there have been few studies estimating the localized concentrations of enzymes within signaling complexes.

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