Table 1.
MS patients: clinical synopsis
Patient no.Sample no.Gender/ageClinical diagnosisLesion type
HSB-2407     F/58 Normal Control 
HSB-2803     M/92 Dementiaa Control 
HSB-3119     F/77 MS diagnosed but not supported by neuropathological diagnosis Control 
HSB-3391     F/48 MS; periventricular white matter shows variable demyelination, associated with prominent gliosis and perivascular lymphocytic and macrophage infiltration Chronic Active MS 
HSB-3442     F/63 Irregular bilateral periventricular demyelination Chronic MS 
HSB-3704     F/47 Extensive white matter demyelination and axonal loss, perivascular lymphocytic cuffing, and extensive gliosis associated with areas of neuronal loss Chronic Active MS 
Patient no.Sample no.Gender/ageClinical diagnosisLesion type
HSB-2407     F/58 Normal Control 
HSB-2803     M/92 Dementiaa Control 
HSB-3119     F/77 MS diagnosed but not supported by neuropathological diagnosis Control 
HSB-3391     F/48 MS; periventricular white matter shows variable demyelination, associated with prominent gliosis and perivascular lymphocytic and macrophage infiltration Chronic Active MS 
HSB-3442     F/63 Irregular bilateral periventricular demyelination Chronic MS 
HSB-3704     F/47 Extensive white matter demyelination and axonal loss, perivascular lymphocytic cuffing, and extensive gliosis associated with areas of neuronal loss Chronic Active MS 

The clinical pathology of MS and control brain tissues obtained from the UCLA HBSFRC is described. Each brain was evaluated by neuropathologists at UCLA. Brain tissue from three MS cases was examined along with three control brains from cases without MS. In all cases, the tissue was taken from the region around or at the corpus callosum, as shown in Fig. S2.

a

Dementia with no distinctive pathology.

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