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In response to salt depletion, aldosterone drives the movement of Na channel protein from ER through the Golgi to the apical membrane in kidney epithelial cells.

Researchers studied the mechanism by which the anticonvulsant Retigabine enhances the activity of K+ channels and found that the drug stabilizes on open state at resting membrane potentials.

In Special Collection: Exocytosis and Endocytosis 2018

Researchers have measured the ability of nerve terminals to buffer Ca2+ entering in response to electrical activity to better understand plasticity of hormone release.

Systematic imaging studies and modeling reveal new details of the regulation of the Gq-coupled GPCR, protease-activated receptor 2, by phosphorylation and β-arrestin.

Voltage-dependent gap junction gating contributes to reverberation in neuronal circuits.


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