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Journal Articles

TCR binding differs for a bacterial superantigen (SEE) and a viral superantigen (Mtv-9).

L Liao, A Marinescu, A Molano, C Ciurli, R P Sekaly, J D Fraser, A Popowicz, D N Posnett

J Exp Med (1996) 184 (4): 1471–1482.

https://doi.org/10.1084/jem.184.4.1471

Published: 01 October 1996

View article titled, TCR binding differs for a bacterial superantigen (SEE) and a viral superantigen (Mtv-9).

Journal Articles

An Epstein-Barr virus-associated superantigen.

N Sutkowski, T Palkama, C Ciurli, R P Sekaly, D A Thorley-Lawson, B T Huber

J Exp Med (1996) 184 (3): 971–980.

https://doi.org/10.1084/jem.184.3.971

Published: 01 September 1996

View article titled, An Epstein-Barr virus-associated superantigen.

Journal Articles

Cross-linking of major histocompatibility complex class II molecules by staphylococcal enterotoxin A superantigen is a requirement for inflammatory cytokine gene expression.

K Mehindate, J Thibodeau, M Dohlsten, T Kalland, R P Sékaly, W Mourad

J Exp Med (1995) 182 (5): 1573–1577.

https://doi.org/10.1084/jem.182.5.1573

Published: 01 November 1995

View article titled, Cross-linking of major histocompatibility complex class II molecules by staphylococcal enterotoxin A superantigen is a requirement for inflammatory cytokine gene expression.

Journal Articles

Identification of residues in the V domain of CD80 (B7-1) implicated in functional interactions with CD28 and CTLA4.

C A Fargeas, A Truneh, M Reddy, M Hurle, R Sweet, R P Sékaly

J Exp Med (1995) 182 (3): 667–675.

https://doi.org/10.1084/jem.182.3.667

Published: 01 September 1995

View article titled, Identification of residues in the V domain of CD80 (B7-1) implicated in functional interactions with CD28 and CTLA4.

Journal Articles

HLA-DR polymorphism affects the interaction with CD4.

S Fleury, J Thibodeau, G Croteau, N Labrecque, H E Aronson, C Cantin, E O Long, R P Sékaly

J Exp Med (1995) 182 (3): 733–741.

https://doi.org/10.1084/jem.182.3.733

Published: 01 September 1995

View article titled, HLA-DR polymorphism affects the interaction with CD4.

Journal Articles

T cell receptor-major histocompatibility complex class II interaction is required for the T cell response to bacterial superantigens.

N Labrecque, J Thibodeau, W Mourad, R P Sékaly

J Exp Med (1994) 180 (5): 1921–1929.

https://doi.org/10.1084/jem.180.5.1921

Published: 01 November 1994

View article titled, T cell receptor-major histocompatibility complex class II interaction is required for the T cell response to bacterial superantigens.

Journal Articles

CD4-mediated enhancement or inhibition of T cell activation does not require the CD4:p56lck association.

A C Zerbib, A B Reske-Kunz, P Lock, R P Sékaly

J Exp Med (1994) 179 (6): 1973–1983.

https://doi.org/10.1084/jem.179.6.1973

Published: 01 June 1994

View article titled, CD4-mediated enhancement or inhibition of T cell activation does not require the CD4:p56lck association.

Journal Articles

Binding sites for bacterial and endogenous retroviral superantigens can be dissociated on major histocompatibility complex class II molecules.

J Thibodeau, N Labrecque, F Denis, B T Huber, R P Sékaly

J Exp Med (1994) 179 (3): 1029–1034.

https://doi.org/10.1084/jem.179.3.1029

Published: 01 March 1994

View article titled, Binding sites for bacterial and endogenous retroviral superantigens can be dissociated on major histocompatibility complex class II molecules.

Journal Articles

Human T cells respond to mouse mammary tumor virus-encoded superantigen: V beta restriction and conserved evolutionary features.

N Labrecque, H McGrath, M Subramanyam, B T Huber, R P Sékaly

J Exp Med (1993) 177 (6): 1735–1743.

https://doi.org/10.1084/jem.177.6.1735

Published: 01 June 1993

View article titled, Human T cells respond to mouse mammary tumor virus-encoded superantigen: V beta restriction and conserved evolutionary features.

Journal Articles

Crosslinking CD4 by human immunodeficiency virus gp120 primes T cells for activation-induced apoptosis.

N K Banda, J Bernier, D K Kurahara, R Kurrle, N Haigwood, R P Sekaly, T H Finkel

J Exp Med (1992) 176 (4): 1099–1106.

https://doi.org/10.1084/jem.176.4.1099

Published: 01 October 1992

View article titled, Crosslinking CD4 by human immunodeficiency virus gp120 primes T cells for activation-induced apoptosis.

Journal Articles

The CD4 molecule is not always required for the T cell response to bacterial enterotoxins.

R P Sékaly, G Croteau, M Bowman, P Scholl, S Burakoff, R S Geha

J Exp Med (1991) 173 (2): 367–371.

https://doi.org/10.1084/jem.173.2.367

Published: 01 February 1991

View article titled, The CD4 molecule is not always required for the T cell response to bacterial enterotoxins.

Journal Articles

HLA-DR alleles differ in their ability to present staphylococcal enterotoxins to T cells.

A Herman, G Croteau, R P Sekaly, J Kappler, P Marrack

J Exp Med (1990) 172 (3): 709–717.

https://doi.org/10.1084/jem.172.3.709

Published: 01 September 1990

View article titled, HLA-DR alleles differ in their ability to present staphylococcal enterotoxins to T cells.

Journal Articles

Complement receptor 2 mediates enhancement of human immunodeficiency virus 1 infection in Epstein-Barr virus-carrying B cells.

M Tremblay, S Meloche, R P Sekaly, M A Wainberg

J Exp Med (1990) 171 (5): 1791–1796.

https://doi.org/10.1084/jem.171.5.1791

Published: 01 May 1990

View article titled, Complement receptor 2 mediates enhancement of human immunodeficiency virus 1 infection in Epstein-Barr virus-carrying B cells.

Journal Articles

Characterization of CD3+, CD4-, CD8- clones expressing the putative T cell receptor gamma gene product. Analysis of the activation pathways leading to interleukin 2 production and triggering of the lytic machinery.

S Ferrini, C Bottino, R Biassoni, A Poggi, R P Sekaly, L Moretta, A Moretta

J Exp Med (1987) 166 (1): 277–282.

https://doi.org/10.1084/jem.166.1.277

Published: 01 July 1987

View article titled, Characterization of CD3+, CD4-, CD8- clones expressing the putative T cell receptor gamma gene product. Analysis of the activation pathways leading to interleukin 2 production and triggering of the lytic machinery.

Journal Articles

Cell surface expression of class II histocompatibility antigens occurs in the absence of the invariant chain.

R P Sekaly, C Tonnelle, M Strubin, B Mach, E O Long

J Exp Med (1986) 164 (5): 1490–1504.

https://doi.org/10.1084/jem.164.5.1490

Published: 01 November 1986

View article titled, Cell surface expression of class II histocompatibility antigens occurs in the absence of the invariant chain.

Journal Articles

Correlated expression of T cell growth factor dependence, sensitivity to Vicia villosa lectin, and cytolytic activity in hybrids between cytolytic T cells and T lymphomas.

A Conzelmann, A Silva, M Cianfriglia, C Tougne, R P Sekaly, M Nabholz

J Exp Med (1982) 156 (5): 1335–1351.

https://doi.org/10.1084/jem.156.5.1335

Published: 01 November 1982

View article titled, Correlated expression of T cell growth factor dependence, sensitivity to Vicia villosa lectin, and cytolytic activity in hybrids between cytolytic T cells and T lymphomas.

Journal Articles

Phenotypic characterization of human cytolytic T lymphocytes in mixed lymphocyte culture.

A Moretta, M C Mingari, B F Haynes, R P Sekaly, L Moretta, A S Fauci

J Exp Med (1981) 153 (1): 213–218.

https://doi.org/10.1084/jem.153.1.213

Published: 01 January 1981

View article titled, Phenotypic characterization of human cytolytic T lymphocytes in mixed lymphocyte culture.

NARROW

TCR binding differs for a bacterial superantigen (SEE) and a viral superantigen (Mtv-9).

An Epstein-Barr virus-associated superantigen.

Cross-linking of major histocompatibility complex class II molecules by staphylococcal enterotoxin A superantigen is a requirement for inflammatory cytokine gene expression.

Identification of residues in the V domain of CD80 (B7-1) implicated in functional interactions with CD28 and CTLA4.

HLA-DR polymorphism affects the interaction with CD4.

T cell receptor-major histocompatibility complex class II interaction is required for the T cell response to bacterial superantigens.

CD4-mediated enhancement or inhibition of T cell activation does not require the CD4:p56lck association.

Binding sites for bacterial and endogenous retroviral superantigens can be dissociated on major histocompatibility complex class II molecules.

Human T cells respond to mouse mammary tumor virus-encoded superantigen: V beta restriction and conserved evolutionary features.

Crosslinking CD4 by human immunodeficiency virus gp120 primes T cells for activation-induced apoptosis.

The CD4 molecule is not always required for the T cell response to bacterial enterotoxins.

HLA-DR alleles differ in their ability to present staphylococcal enterotoxins to T cells.

Complement receptor 2 mediates enhancement of human immunodeficiency virus 1 infection in Epstein-Barr virus-carrying B cells.

Characterization of CD3+, CD4-, CD8- clones expressing the putative T cell receptor gamma gene product. Analysis of the activation pathways leading to interleukin 2 production and triggering of the lytic machinery.

Cell surface expression of class II histocompatibility antigens occurs in the absence of the invariant chain.

Correlated expression of T cell growth factor dependence, sensitivity to Vicia villosa lectin, and cytolytic activity in hybrids between cytolytic T cells and T lymphomas.

Phenotypic characterization of human cytolytic T lymphocytes in mixed lymphocyte culture.

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