Search Results | Journal of Experimental Medicine

Skip Nav Destination

1-6 of 6

Sort by

Sort Order Select

Journal Articles

IL-1β mediates chronic intestinal inflammation by promoting the accumulation of IL-17A secreting innate lymphoid cells and CD4⁺ Th17 cells

Margherita Coccia, Oliver J. Harrison, Chris Schiering, Mark J. Asquith, Burkhard Becher, Fiona Powrie, Kevin J. Maloy

J Exp Med (2012) 209 (9): 1595–1609.

DOI: https://doi.org/10.1084/jem.20111453

Published: 13 August 2012

View article

Journal Articles

Identification of a genetic locus controlling bacteria-driven colitis and associated cancer through effects on innate inflammation

Olivier Boulard, Stefanie Kirchberger, Daniel J. Royston, Kevin J. Maloy, Fiona M. Powrie

J Exp Med (2012) 209 (7): 1309–1324.

DOI: https://doi.org/10.1084/jem.20120239

Published: 25 June 2012

View article

Includes: Supplementary data

Journal Articles

Interleukin-23 drives innate and T cell–mediated intestinal inflammation

Sophie Hue, Philip Ahern, Sofia Buonocore, Marika C. Kullberg, Daniel J. Cua, Brent S. McKenzie, Fiona Powrie, Kevin J. Maloy

J Exp Med (2006) 203 (11): 2473–2483.

DOI: https://doi.org/10.1084/jem.20061099

Published: 09 October 2006

View article

Journal Articles

IL-23 plays a key role in Helicobacter hepaticus–induced T cell–dependent colitis

Marika C. Kullberg, Dragana Jankovic, Carl G. Feng, Sophie Hue, Peter L. Gorelick, Brent S. McKenzie, Daniel J. Cua, Fiona Powrie, Allen W. Cheever, Kevin J. Maloy, Alan Sher

J Exp Med (2006) 203 (11): 2485–2494.

DOI: https://doi.org/10.1084/jem.20061082

Published: 09 October 2006

View article

Journal Articles

CD4⁺CD25⁺ T_R Cells Suppress Innate Immune Pathology Through Cytokine-dependent Mechanisms

Kevin J. Maloy, Laurence Salaun, Rachel Cahill, Gordon Dougan, Nigel J. Saunders, Fiona Powrie

J Exp Med (2003) 197 (1): 111–119.

DOI: https://doi.org/10.1084/jem.20021345

Published: 06 January 2003

View article

Journal Articles

Cd4⁺ T Cell Subsets during Virus Infection: Protective Capacity Depends on Effector Cytokine Secretion and on Migratory Capability

Kevin J. Maloy, Christoph Burkhart, Tobias M. Junt, Bernhard Odermatt, Annette Oxenius, Luca Piali, Rolf M. Zinkernagel, Hans Hengartner

J Exp Med (2000) 191 (12): 2159–2170.

DOI: https://doi.org/10.1084/jem.191.12.2159

Published: 19 June 2000

View article

NARROW

IL-1β mediates chronic intestinal inflammation by promoting the accumulation of IL-17A secreting innate lymphoid cells and CD4⁺ Th17 cells

Identification of a genetic locus controlling bacteria-driven colitis and associated cancer through effects on innate inflammation

Interleukin-23 drives innate and T cell–mediated intestinal inflammation

IL-23 plays a key role in Helicobacter hepaticus–induced T cell–dependent colitis

CD4⁺CD25⁺ T_R Cells Suppress Innate Immune Pathology Through Cytokine-dependent Mechanisms

Cd4⁺ T Cell Subsets during Virus Infection: Protective Capacity Depends on Effector Cytokine Secretion and on Migratory Capability

Email alerts

IL-1β mediates chronic intestinal inflammation by promoting the accumulation of IL-17A secreting innate lymphoid cells and CD4+ Th17 cells

Identification of a genetic locus controlling bacteria-driven colitis and associated cancer through effects on innate inflammation

Interleukin-23 drives innate and T cell–mediated intestinal inflammation

IL-23 plays a key role in Helicobacter hepaticus–induced T cell–dependent colitis

CD4+CD25+ TR Cells Suppress Innate Immune Pathology Through Cytokine-dependent Mechanisms

Cd4+ T Cell Subsets during Virus Infection: Protective Capacity Depends on Effector Cytokine Secretion and on Migratory Capability

Email alerts

This Feature Is Available To Subscribers Only

IL-1β mediates chronic intestinal inflammation by promoting the accumulation of IL-17A secreting innate lymphoid cells and CD4⁺ Th17 cells

CD4⁺CD25⁺ T_R Cells Suppress Innate Immune Pathology Through Cytokine-dependent Mechanisms

Cd4⁺ T Cell Subsets during Virus Infection: Protective Capacity Depends on Effector Cytokine Secretion and on Migratory Capability