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Journal Articles

Signaling capacity of the T cell antigen receptor is negatively regulated by the PTP1C tyrosine phosphatase.

G Pani, K D Fischer, I Mlinaric-Rascan, K A Siminovitch

J Exp Med (1996) 184 (3): 839–852.

https://doi.org/10.1084/jem.184.3.839

Published: 01 September 1996

View article titled, Signaling capacity of the T cell antigen receptor is negatively regulated by the PTP1C tyrosine phosphatase.

Journal Articles

Identification of the tyrosine phosphatase PTP1C as a B cell antigen receptor-associated protein involved in the regulation of B cell signaling.

G Pani, M Kozlowski, J C Cambier, G B Mills, K A Siminovitch

J Exp Med (1995) 181 (6): 2077–2084.

https://doi.org/10.1084/jem.181.6.2077

Published: 01 June 1995

View article titled, Identification of the tyrosine phosphatase PTP1C as a B cell antigen receptor-associated protein involved in the regulation of B cell signaling.

Journal Articles

Expression and catalytic activity of the tyrosine phosphatase PTP1C is severely impaired in motheaten and viable motheaten mice.

M Kozlowski, I Mlinaric-Rascan, G S Feng, R Shen, T Pawson, K A Siminovitch

J Exp Med (1993) 178 (6): 2157–2163.

https://doi.org/10.1084/jem.178.6.2157

Published: 01 December 1993

View article titled, Expression and catalytic activity of the tyrosine phosphatase PTP1C is severely impaired in motheaten and viable motheaten mice.

Journal Articles

Reduction of lupus nephritis in MRL/lpr mice by a bacterial superantigen treatment.

C Kim, K A Siminovitch, A Ochi

J Exp Med (1991) 174 (6): 1431–1437.

https://doi.org/10.1084/jem.174.6.1431

Published: 01 December 1991

View article titled, Reduction of lupus nephritis in MRL/lpr mice by a bacterial superantigen treatment.

Journal Articles

An expanded population of natural killer cells in mice with severe combined immunodeficiency (SCID) lack rearrangement and expression of T cell receptor genes.

R J Lauzon, K A Siminovitch, G M Fulop, R A Phillips, J C Roder

J Exp Med (1986) 164 (5): 1797–1802.

https://doi.org/10.1084/jem.164.5.1797

Published: 01 November 1986

View article titled, An expanded population of natural killer cells in mice with severe combined immunodeficiency (SCID) lack rearrangement and expression of T cell receptor genes.

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Signaling capacity of the T cell antigen receptor is negatively regulated by the PTP1C tyrosine phosphatase.

Identification of the tyrosine phosphatase PTP1C as a B cell antigen receptor-associated protein involved in the regulation of B cell signaling.

Expression and catalytic activity of the tyrosine phosphatase PTP1C is severely impaired in motheaten and viable motheaten mice.

Reduction of lupus nephritis in MRL/lpr mice by a bacterial superantigen treatment.

An expanded population of natural killer cells in mice with severe combined immunodeficiency (SCID) lack rearrangement and expression of T cell receptor genes.

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Signaling capacity of the T cell antigen receptor is negatively regulated by the PTP1C tyrosine phosphatase.

Identification of the tyrosine phosphatase PTP1C as a B cell antigen receptor-associated protein involved in the regulation of B cell signaling.

Expression and catalytic activity of the tyrosine phosphatase PTP1C is severely impaired in motheaten and viable motheaten mice.

Reduction of lupus nephritis in MRL/lpr mice by a bacterial superantigen treatment.

An expanded population of natural killer cells in mice with severe combined immunodeficiency (SCID) lack rearrangement and expression of T cell receptor genes.

Email alerts

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