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Tumor Necrosis Factor Sustains the Generalized Lymphoproliferative Disorder ( gld ) Phenotype

Heinrich Körner, Erika Cretney, Patricia Wilhelm, Janice M. Kelly, Martin Röllinghoff, Jonathon D. Sedgwick, Mark J. Smyth

J Exp Med (2000) 191 (1): 89–96.

https://doi.org/10.1084/jem.191.1.89

Published: 03 January 2000

View article titled, Tumor Necrosis Factor Sustains the Generalized Lymphoproliferative Disorder ( <em>gld</em> ) Phenotype

Journal Articles

Lymphotoxin α/β and Tumor Necrosis Factor Are Required for Stromal Cell Expression of Homing Chemokines in B and T Cell Areas of the Spleen

Vu N. Ngo, Heinrich Korner, Michael D. Gunn, Kerstin N. Schmidt, D. Sean Riminton, Max D. Cooper, Jeffrey L. Browning, Jonathon D. Sedgwick, Jason G. Cyster

J Exp Med (1999) 189 (2): 403–412.

https://doi.org/10.1084/jem.189.2.403

Published: 18 January 1999

View article titled, Lymphotoxin α/β and Tumor Necrosis Factor Are Required for Stromal Cell Expression of Homing Chemokines in B and T Cell Areas of the Spleen

Journal Articles

An Essential Role for Tumor Necrosis Factor in Natural Killer Cell–mediated Tumor Rejection in the Peritoneum

Mark J. Smyth, Janice M. Kelly, Alan G. Baxter, Heinrich Körner, Jonathon D. Sedgwick

J Exp Med (1998) 188 (9): 1611–1619.

https://doi.org/10.1084/jem.188.9.1611

Published: 02 November 1998

View article titled, An Essential Role for Tumor Necrosis Factor in Natural Killer Cell–mediated Tumor Rejection in the Peritoneum

Journal Articles

Generation of Splenic Follicular Structure and B Cell Movement in Tumor Necrosis Factor–deficient Mice

Matthew C. Cook, Heinrich Körner, D. Sean Riminton, Frances A. Lemckert, Jhagvaral Hasbold, Michelle Amesbury, Philip D. Hodgkin, Jason G. Cyster, Jonathon D. Sedgwick, Antony Basten

J Exp Med (1998) 188 (8): 1503–1510.

https://doi.org/10.1084/jem.188.8.1503

Published: 19 October 1998

View article titled, Generation of Splenic Follicular Structure and B Cell Movement in Tumor Necrosis Factor–deficient Mice

Journal Articles

Challenging Cytokine Redundancy: Inflammatory Cell Movement and Clinical Course of Experimental Autoimmune Encephalomyelitis Are Normal in Lymphotoxin-deficient, but Not Tumor Necrosis Factor–deficient, Mice

D. Sean Riminton, Heinrich Körner, Deborah H. Strickland, Frances A. Lemckert, John D. Pollard, Jonathon D. Sedgwick

J Exp Med (1998) 187 (9): 1517–1528.

https://doi.org/10.1084/jem.187.9.1517

Published: 04 May 1998

View article titled, Challenging Cytokine Redundancy: Inflammatory Cell Movement and Clinical Course of Experimental Autoimmune Encephalomyelitis Are Normal in Lymphotoxin-deficient, but Not Tumor Necrosis Factor–deficient, Mice

Journal Articles

Critical Points of Tumor Necrosis Factor Action in Central Nervous System Autoimmune Inflammation Defined by Gene Targeting

Heinrich Körner, D. Sean Riminton, Deborah H. Strickland, Frances A. Lemckert, John D. Pollard, Jonathon D. Sedgwick

J Exp Med (1997) 186 (9): 1585–1590.

https://doi.org/10.1084/jem.186.9.1585

Published: 03 November 1997

View article titled, Critical Points of Tumor Necrosis Factor Action in Central Nervous System Autoimmune Inflammation Defined by Gene Targeting

NARROW

Tumor Necrosis Factor Sustains the Generalized Lymphoproliferative Disorder ( gld ) Phenotype

Lymphotoxin α/β and Tumor Necrosis Factor Are Required for Stromal Cell Expression of Homing Chemokines in B and T Cell Areas of the Spleen

An Essential Role for Tumor Necrosis Factor in Natural Killer Cell–mediated Tumor Rejection in the Peritoneum

Generation of Splenic Follicular Structure and B Cell Movement in Tumor Necrosis Factor–deficient Mice

Challenging Cytokine Redundancy: Inflammatory Cell Movement and Clinical Course of Experimental Autoimmune Encephalomyelitis Are Normal in Lymphotoxin-deficient, but Not Tumor Necrosis Factor–deficient, Mice

Critical Points of Tumor Necrosis Factor Action in Central Nervous System Autoimmune Inflammation Defined by Gene Targeting

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Tumor Necrosis Factor Sustains the Generalized Lymphoproliferative Disorder ( gld ) Phenotype

Lymphotoxin α/β and Tumor Necrosis Factor Are Required for Stromal Cell Expression of Homing Chemokines in B and T Cell Areas of the Spleen

An Essential Role for Tumor Necrosis Factor in Natural Killer Cell–mediated Tumor Rejection in the Peritoneum

Generation of Splenic Follicular Structure and B Cell Movement in Tumor Necrosis Factor–deficient Mice

Challenging Cytokine Redundancy: Inflammatory Cell Movement and Clinical Course of Experimental Autoimmune Encephalomyelitis Are Normal in Lymphotoxin-deficient, but Not Tumor Necrosis Factor–deficient, Mice

Critical Points of Tumor Necrosis Factor Action in Central Nervous System Autoimmune Inflammation Defined by Gene Targeting

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