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Journal Articles

Inhibition of tumor growth by elimination of granulocytes.

L A Pekarek, B A Starr, A Y Toledano, H Schreiber

J Exp Med (1995) 181 (1): 435–440.

https://doi.org/10.1084/jem.181.1.435

Published: 01 January 1995

View article titled, Inhibition of tumor growth by elimination of granulocytes.

Journal Articles

A tumor escape variant that has lost one major histocompatibility complex class I restriction element induces specific CD8+ T cells to an antigen that no longer serves as a target.

S Seung, J L Urban, H Schreiber

J Exp Med (1993) 178 (3): 933–940.

https://doi.org/10.1084/jem.178.3.933

Published: 01 September 1993

View article titled, A tumor escape variant that has lost one major histocompatibility complex class I restriction element induces specific CD8+ T cells to an antigen that no longer serves as a target.

Journal Articles

Stroma is critical for preventing or permitting immunological destruction of antigenic cancer cells.

S Singh, S R Ross, M Acena, D A Rowley, H Schreiber

J Exp Med (1992) 175 (1): 139–146.

https://doi.org/10.1084/jem.175.1.139

Published: 01 January 1992

View article titled, Stroma is critical for preventing or permitting immunological destruction of antigenic cancer cells.

Journal Articles

Animals bearing malignant grafts reject normal grafts that express through gene transfer the same antigen.

G A Perdrizet, S R Ross, H J Stauss, S Singh, H Koeppen, H Schreiber

J Exp Med (1990) 171 (4): 1205–1220.

https://doi.org/10.1084/jem.171.4.1205

Published: 01 April 1990

View article titled, Animals bearing malignant grafts reject normal grafts that express through gene transfer the same antigen.

Journal Articles

Tumor antigens defined by cloned immunological probes are highly polymorphic and are not detected on autologous normal cells.

P L Ward, H Koeppen, T Hurteau, H Schreiber

J Exp Med (1989) 170 (1): 217–232.

https://doi.org/10.1084/jem.170.1.217

Published: 01 July 1989

View article titled, Tumor antigens defined by cloned immunological probes are highly polymorphic and are not detected on autologous normal cells.

Journal Articles

Tumor necrosis factor/cachectin. Induction of hemorrhagic necrosis in normal tissue requires the fifth component of complement (C5).

J L Rothstein, T F Lint, H Schreiber

J Exp Med (1988) 168 (6): 2007–2021.

https://doi.org/10.1084/jem.168.6.2007

Published: 01 December 1988

View article titled, Tumor necrosis factor/cachectin. Induction of hemorrhagic necrosis in normal tissue requires the fifth component of complement (C5).

Journal Articles

Highly malignant tumor variants retain tumor-specific antigens recognized by T helper cells.

C Van Waes, J L Urban, J L Rothstein, P L Ward, H Schreiber

J Exp Med (1986) 164 (5): 1547–1565.

https://doi.org/10.1084/jem.164.5.1547

Published: 01 November 1986

View article titled, Highly malignant tumor variants retain tumor-specific antigens recognized by T helper cells.

Journal Articles

Identification of a unique tumor antigen as rejection antigen by molecular cloning and gene transfer.

H J Stauss, C Van Waes, M A Fink, B Starr, H Schreiber

J Exp Med (1986) 164 (5): 1516–1530.

https://doi.org/10.1084/jem.164.5.1516

Published: 01 November 1986

View article titled, Identification of a unique tumor antigen as rejection antigen by molecular cloning and gene transfer.

Journal Articles

Multiple cancers. Tumor burden permits the outgrowth of other cancers.

C A Mullen, J L Urban, C Van Waes, D A Rowley, H Schreiber

J Exp Med (1985) 162 (5): 1665–1682.

https://doi.org/10.1084/jem.162.5.1665

Published: 01 November 1985

View article titled, Multiple cancers. Tumor burden permits the outgrowth of other cancers.

Journal Articles

Selection of macrophage-resistant progressor tumor variants by the normal host. Requirement for concomitant T cell-mediated immunity.

J L Urban, H Schreiber

J Exp Med (1983) 157 (2): 642–656.

https://doi.org/10.1084/jem.157.2.642

Published: 01 February 1983

View article titled, Selection of macrophage-resistant progressor tumor variants by the normal host. Requirement for concomitant T cell-mediated immunity.

Journal Articles

Immunoselection of tumor cell variants by mice suppressed with ultraviolet radiation.

J L Urban, J M Holland, M L Kripke, H Schreiber

J Exp Med (1982) 156 (4): 1025–1041.

https://doi.org/10.1084/jem.156.4.1025

Published: 01 October 1982

View article titled, Immunoselection of tumor cell variants by mice suppressed with ultraviolet radiation.

Journal Articles

Mechanisms of syngeneic tumor rejection. Susceptibility of host-selected progressor variants to various immunological effector cells.

J L Urban, R C Burton, J M Holland, M L Kripke, H Schreiber

J Exp Med (1982) 155 (2): 557–573.

https://doi.org/10.1084/jem.155.2.557

Published: 01 February 1982

View article titled, Mechanisms of syngeneic tumor rejection. Susceptibility of host-selected progressor variants to various immunological effector cells.

Journal Articles

Loss of tumor-specific and idiotype-specific immunity with age.

P M Flood, J L Urban, M L Kripke, H Schreiber

J Exp Med (1981) 154 (2): 275–290.

https://doi.org/10.1084/jem.154.2.275

Published: 01 August 1981

View article titled, Loss of tumor-specific and idiotype-specific immunity with age.

Journal Articles

Suppression by autogenous complementary idiotypes: the priority of the first response.

D A Rowley, H Köhler, H Schreiber, S T Kaye, I Lorbach

J Exp Med (1976) 144 (4): 946–959.

https://doi.org/10.1084/jem.144.4.946

Published: 01 October 1976

View article titled, Suppression by autogenous complementary idiotypes: the priority of the first response.

NARROW

Inhibition of tumor growth by elimination of granulocytes.

A tumor escape variant that has lost one major histocompatibility complex class I restriction element induces specific CD8+ T cells to an antigen that no longer serves as a target.

Stroma is critical for preventing or permitting immunological destruction of antigenic cancer cells.

Animals bearing malignant grafts reject normal grafts that express through gene transfer the same antigen.

Tumor antigens defined by cloned immunological probes are highly polymorphic and are not detected on autologous normal cells.

Tumor necrosis factor/cachectin. Induction of hemorrhagic necrosis in normal tissue requires the fifth component of complement (C5).

Highly malignant tumor variants retain tumor-specific antigens recognized by T helper cells.

Identification of a unique tumor antigen as rejection antigen by molecular cloning and gene transfer.

Multiple cancers. Tumor burden permits the outgrowth of other cancers.

Selection of macrophage-resistant progressor tumor variants by the normal host. Requirement for concomitant T cell-mediated immunity.

Immunoselection of tumor cell variants by mice suppressed with ultraviolet radiation.

Mechanisms of syngeneic tumor rejection. Susceptibility of host-selected progressor variants to various immunological effector cells.

Loss of tumor-specific and idiotype-specific immunity with age.

Suppression by autogenous complementary idiotypes: the priority of the first response.

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