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Expansion of somatically reverted memory CD8⁺ T cells in patients with X-linked lymphoproliferative disease caused by selective pressure from Epstein-Barr virus

Umaimainthan Palendira, Carol Low, Andrew I. Bell, Cindy S. Ma, Rachel J.M. Abbott, Tri Giang Phan, D. Sean Riminton, Sharon Choo, Joanne M. Smart, Vassilios Lougaris, Silvia Giliani, Rebecca H. Buckley, Bodo Grimbacher, Frank Alvaro, Amy D. Klion, Kim E. Nichols, Stephen Adelstein, Alan B. Rickinson, Stuart G. Tangye

J Exp Med (2012) 209 (5): 913–924.

https://doi.org/10.1084/jem.20112391

Published: 09 April 2012

View article titled, Expansion of somatically reverted memory CD8<sup>+</sup> T cells in patients with X-linked lymphoproliferative disease caused by selective pressure from Epstein-Barr virus

Journal Articles

B cell–intrinsic signaling through IL-21 receptor and STAT3 is required for establishing long-lived antibody responses in humans

J Exp Med (2010) 207 (1): 155–171.

https://doi.org/10.1084/jem.20091706

Published: 04 January 2010

View article titled, B cell–intrinsic signaling through IL-21 receptor and STAT3 is required for establishing long-lived antibody responses in humans

Includes: Supplementary data

Journal Articles

Lymphotoxin α/β and Tumor Necrosis Factor Are Required for Stromal Cell Expression of Homing Chemokines in B and T Cell Areas of the Spleen

Vu N. Ngo, Heinrich Korner, Michael D. Gunn, Kerstin N. Schmidt, D. Sean Riminton, Max D. Cooper, Jeffrey L. Browning, Jonathon D. Sedgwick, Jason G. Cyster

J Exp Med (1999) 189 (2): 403–412.

https://doi.org/10.1084/jem.189.2.403

Published: 18 January 1999

View article titled, Lymphotoxin α/β and Tumor Necrosis Factor Are Required for Stromal Cell Expression of Homing Chemokines in B and T Cell Areas of the Spleen

Journal Articles

Generation of Splenic Follicular Structure and B Cell Movement in Tumor Necrosis Factor–deficient Mice

Matthew C. Cook, Heinrich Körner, D. Sean Riminton, Frances A. Lemckert, Jhagvaral Hasbold, Michelle Amesbury, Philip D. Hodgkin, Jason G. Cyster, Jonathon D. Sedgwick, Antony Basten

J Exp Med (1998) 188 (8): 1503–1510.

https://doi.org/10.1084/jem.188.8.1503

Published: 19 October 1998

View article titled, Generation of Splenic Follicular Structure and B Cell Movement in Tumor Necrosis Factor–deficient Mice

Journal Articles

Challenging Cytokine Redundancy: Inflammatory Cell Movement and Clinical Course of Experimental Autoimmune Encephalomyelitis Are Normal in Lymphotoxin-deficient, but Not Tumor Necrosis Factor–deficient, Mice

D. Sean Riminton, Heinrich Körner, Deborah H. Strickland, Frances A. Lemckert, John D. Pollard, Jonathon D. Sedgwick

J Exp Med (1998) 187 (9): 1517–1528.

https://doi.org/10.1084/jem.187.9.1517

Published: 04 May 1998

View article titled, Challenging Cytokine Redundancy: Inflammatory Cell Movement and Clinical Course of Experimental Autoimmune Encephalomyelitis Are Normal in Lymphotoxin-deficient, but Not Tumor Necrosis Factor–deficient, Mice

Journal Articles

Critical Points of Tumor Necrosis Factor Action in Central Nervous System Autoimmune Inflammation Defined by Gene Targeting

Heinrich Körner, D. Sean Riminton, Deborah H. Strickland, Frances A. Lemckert, John D. Pollard, Jonathon D. Sedgwick

J Exp Med (1997) 186 (9): 1585–1590.

https://doi.org/10.1084/jem.186.9.1585

Published: 03 November 1997

View article titled, Critical Points of Tumor Necrosis Factor Action in Central Nervous System Autoimmune Inflammation Defined by Gene Targeting

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Expansion of somatically reverted memory CD8⁺ T cells in patients with X-linked lymphoproliferative disease caused by selective pressure from Epstein-Barr virus

B cell–intrinsic signaling through IL-21 receptor and STAT3 is required for establishing long-lived antibody responses in humans

Lymphotoxin α/β and Tumor Necrosis Factor Are Required for Stromal Cell Expression of Homing Chemokines in B and T Cell Areas of the Spleen

Generation of Splenic Follicular Structure and B Cell Movement in Tumor Necrosis Factor–deficient Mice

Challenging Cytokine Redundancy: Inflammatory Cell Movement and Clinical Course of Experimental Autoimmune Encephalomyelitis Are Normal in Lymphotoxin-deficient, but Not Tumor Necrosis Factor–deficient, Mice

Critical Points of Tumor Necrosis Factor Action in Central Nervous System Autoimmune Inflammation Defined by Gene Targeting

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Expansion of somatically reverted memory CD8+ T cells in patients with X-linked lymphoproliferative disease caused by selective pressure from Epstein-Barr virus

B cell–intrinsic signaling through IL-21 receptor and STAT3 is required for establishing long-lived antibody responses in humans

Lymphotoxin α/β and Tumor Necrosis Factor Are Required for Stromal Cell Expression of Homing Chemokines in B and T Cell Areas of the Spleen

Generation of Splenic Follicular Structure and B Cell Movement in Tumor Necrosis Factor–deficient Mice

Challenging Cytokine Redundancy: Inflammatory Cell Movement and Clinical Course of Experimental Autoimmune Encephalomyelitis Are Normal in Lymphotoxin-deficient, but Not Tumor Necrosis Factor–deficient, Mice

Critical Points of Tumor Necrosis Factor Action in Central Nervous System Autoimmune Inflammation Defined by Gene Targeting

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Expansion of somatically reverted memory CD8⁺ T cells in patients with X-linked lymphoproliferative disease caused by selective pressure from Epstein-Barr virus