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Commensal bacteria promote type I interferon signaling to maintain immune tolerance in mice

In Special Collection:

JEM Immunology Collection 2024 , Innate Immune Signaling: From Host Defense to Disease Pathogenesis

Adriana Vasquez Ayala, Chia-Yun Hsu, Renee E. Oles, Kazuhiko Matsuo, Luke R. Loomis, Ekaterina Buzun, Marvic Carrillo Terrazas, Romana R. Gerner, Hsueh-Han Lu, Sohee Kim, Ziyue Zhang, Jong Hwee Park, Paul Rivaud, Matt Thomson, Li-Fan Lu, Booki Min, Hiutung Chu

J Exp Med (2023) 221 (1): e20230063.

https://doi.org/10.1084/jem.20230063

Published: 12 December 2023

View article titled, Commensal bacteria promote type I interferon signaling to maintain immune tolerance in mice

Journal Articles

Gut epithelial IL-27 confers intestinal immunity through the induction of intraepithelial lymphocytes

Chia-Hao Lin, Mei-Chi Chen, Ling-Li Lin, David A. Christian, Booki Min, Christopher A. Hunter, Li-Fan Lu

J Exp Med (2021) 218 (11): e20210021.

https://doi.org/10.1084/jem.20210021

Published: 23 September 2021

View article titled, Gut epithelial IL-27 confers intestinal immunity through the induction of intraepithelial lymphocytes

Journal Articles

A critical role for IRAK4 kinase activity in Toll-like receptor–mediated innate immunity

Tae Whan Kim, Kirk Staschke, Katarzyna Bulek, Jianhong Yao, Kristi Peters, Keun-Hee Oh, Yvonne Vandenburg, Hui Xiao, Wen Qian, Tom Hamilton, Booki Min, Ganes Sen, Raymond Gilmour, Xiaoxia Li

J Exp Med (2007) 204 (5): 1025–1036.

https://doi.org/10.1084/jem.20061825

Published: 30 April 2007

View article titled, A critical role for IRAK4 kinase activity in Toll-like receptor–mediated innate immunity

Journal Articles

Basophils Produce IL-4 and Accumulate in Tissues after Infection with a Th2-inducing Parasite

Booki Min, Melanie Prout, Jane Hu-Li, Jinfang Zhu, Dragana Jankovic, Ellen S. Morgan, Joseph F. Urban, Jr., Ann M. Dvorak, Fred D. Finkelman, Graham LeGros, William E. Paul

J Exp Med (2004) 200 (4): 507–517.

https://doi.org/10.1084/jem.20040590

Published: 16 August 2004

View article titled, Basophils Produce IL-4 and Accumulate in Tissues after Infection with a Th2-inducing Parasite

Includes: Supplementary data

Journal Articles

Nonredundant Roles for CD1d-restricted Natural Killer T Cells and Conventional CD4⁺ T Cells in the Induction of Immunoglobulin E Antibodies in Response to Interleukin 18 Treatment of Mice

Tomohiro Yoshimoto, Booki Min, Takaaki Sugimoto, Nobuki Hayashi, Yuriko Ishikawa, Yuki Sasaki, Hitomi Hata, Kazuyoshi Takeda, Ko Okumura, Luc Van Kaer, William E. Paul, Kenji Nakanishi

J Exp Med (2003) 197 (8): 997–1005.

https://doi.org/10.1084/jem.20021701

Published: 14 April 2003

View article titled, Nonredundant Roles for CD1d-restricted Natural Killer T Cells and Conventional CD4<sup>+</sup> T Cells in the Induction of Immunoglobulin E Antibodies in Response to Interleukin 18 Treatment of Mice

Journal Articles

Coupling of Peripheral Tolerance to Endogenous Interleukin 10 Promotes Effective Modulation of Myelin-Activated T Cells and Ameliorates Experimental Allergic Encephalomyelitis

Kevin L. Legge, Booki Min, J. Jeremiah Bell, Jacque C. Caprio, Lequn Li, Randal K. Gregg, Habib Zaghouani

J Exp Med (2000) 191 (12): 2039–2052.

https://doi.org/10.1084/jem.191.12.2039

Published: 12 June 2000

View article titled, Coupling of Peripheral Tolerance to Endogenous Interleukin 10 Promotes Effective Modulation of Myelin-Activated T Cells and Ameliorates Experimental Allergic Encephalomyelitis

Journal Articles

Neonatal Exposure to a Self-Peptide–Immunoglobulin Chimera Circumvents the Use of Adjuvant and Confers Resistance to Autoimmune Disease by a Novel Mechanism Involving Interleukin 4 Lymph Node Deviation and Interferon γ–mediated Splenic Anergy

Booki Min, Kevin L. Legge, Christopher Pack, Habib Zaghouani

J Exp Med (1998) 188 (11): 2007–2017.

https://doi.org/10.1084/jem.188.11.2007

Published: 07 December 1998

View article titled, Neonatal Exposure to a Self-Peptide–Immunoglobulin Chimera Circumvents the Use of Adjuvant and Confers Resistance to Autoimmune Disease by a Novel Mechanism Involving Interleukin 4 Lymph Node Deviation and Interferon γ–mediated Splenic Anergy

Journal Articles

Presentation of a T Cell Receptor Antagonist Peptide by Immunoglobulins Ablates Activation of T Cells by a Synthetic Peptide or Proteins Requiring Endocytic Processing

Kevin L. Legge, Booki Min, Nicholas T. Potter, Habib Zaghouani

J Exp Med (1997) 185 (6): 1043–1054.

https://doi.org/10.1084/jem.185.6.1043

Published: 17 March 1997

View article titled, Presentation of a T Cell Receptor Antagonist Peptide by Immunoglobulins Ablates Activation of T Cells by a Synthetic Peptide or Proteins Requiring Endocytic Processing

NARROW

Commensal bacteria promote type I interferon signaling to maintain immune tolerance in mice

Gut epithelial IL-27 confers intestinal immunity through the induction of intraepithelial lymphocytes

A critical role for IRAK4 kinase activity in Toll-like receptor–mediated innate immunity

Basophils Produce IL-4 and Accumulate in Tissues after Infection with a Th2-inducing Parasite

Nonredundant Roles for CD1d-restricted Natural Killer T Cells and Conventional CD4⁺ T Cells in the Induction of Immunoglobulin E Antibodies in Response to Interleukin 18 Treatment of Mice

Coupling of Peripheral Tolerance to Endogenous Interleukin 10 Promotes Effective Modulation of Myelin-Activated T Cells and Ameliorates Experimental Allergic Encephalomyelitis

Neonatal Exposure to a Self-Peptide–Immunoglobulin Chimera Circumvents the Use of Adjuvant and Confers Resistance to Autoimmune Disease by a Novel Mechanism Involving Interleukin 4 Lymph Node Deviation and Interferon γ–mediated Splenic Anergy

Presentation of a T Cell Receptor Antagonist Peptide by Immunoglobulins Ablates Activation of T Cells by a Synthetic Peptide or Proteins Requiring Endocytic Processing

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