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A G Rolink
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Journal Articles

Intrinsic B cell defects in NZB and NZW mice contribute to systemic lupus erythematosus in (NZB x NZW)F1 mice.

L Reininger, T H Winkler, C P Kalberer, M Jourdan, F Melchers, A G Rolink
Journal: Journal of Experimental Medicine
J Exp Med (1996) 184 (3): 853–861.
https://doi.org/10.1084/jem.184.3.853
Published: 01 September 1996
View article titled, Intrinsic B cell defects in NZB and NZW mice contribute to systemic lupus erythematosus in (NZB x NZW)F1 mice.
Open the PDF for Intrinsic B cell defects in NZB and NZW mice contribute to systemic lupus erythematosus in (NZB x NZW)F1 mice. in another window
Journal Articles

Development of autoimmune disease in SCID mice populated with long-term "in vitro" proliferating (NZB x NZW)F1 pre-B cells.

L Reininger, T Radaszkiewicz, M Kosco, F Melchers, A G Rolink
Journal: Journal of Experimental Medicine
J Exp Med (1992) 176 (5): 1343–1353.
https://doi.org/10.1084/jem.176.5.1343
Published: 01 November 1992
View article titled, Development of autoimmune disease in SCID mice populated with long-term "in vitro" proliferating (NZB x NZW)F1 pre-B cells.
Open the PDF for Development of autoimmune disease in SCID mice populated with long-term "in vitro" proliferating (NZB x NZW)F1 pre-B cells. in another window
Journal Articles

Monoclonal antibodies reactive with the mouse interleukin 5 receptor.

A G Rolink, F Melchers, R Palacios
Journal: Journal of Experimental Medicine
J Exp Med (1989) 169 (5): 1693–1701.
https://doi.org/10.1084/jem.169.5.1693
Published: 01 May 1989
View article titled, Monoclonal antibodies reactive with the mouse interleukin 5 receptor.
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Journal Articles

The autoantigen-binding B cell repertoires of normal and of chronically graft-versus-host-diseased mice.

A G Rolink, T Radaszkiewicz, F Melchers
Journal: Journal of Experimental Medicine
J Exp Med (1987) 165 (6): 1675–1687.
https://doi.org/10.1084/jem.165.6.1675
Published: 01 June 1987
View article titled, The autoantigen-binding B cell repertoires of normal and of chronically graft-versus-host-diseased mice.
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Journal Articles

Allosuppressor- and allohelper-T cells in acute and chronic graft-vs.-host (GVH) disease. III. Different Lyt subsets of donor T cells induce different pathological syndromes.

A G Rolink, E Gleichmann
Journal: Journal of Experimental Medicine
J Exp Med (1983) 158 (2): 546–558.
https://doi.org/10.1084/jem.158.2.546
Published: 01 August 1983
View article titled, Allosuppressor- and allohelper-T cells in acute and chronic graft-vs.-host (GVH) disease. III. Different Lyt subsets of donor T cells induce different pathological syndromes.
Open the PDF for Allosuppressor- and allohelper-T cells in acute and chronic graft-vs.-host (GVH) disease. III. Different Lyt subsets of donor T cells induce different pathological syndromes. in another window
Journal Articles

Allosuppressor and allohelper T cells in acute and chronic graft-vs.-host disease. II. F1 recipients carrying mutations at H-2K and/or I-A.

A G Rolink, S T Pals, E Gleichmann
Journal: Journal of Experimental Medicine
J Exp Med (1983) 157 (2): 755–771.
https://doi.org/10.1084/jem.157.2.755
Published: 01 February 1983
View article titled, Allosuppressor and allohelper T cells in acute and chronic graft-vs.-host disease. II. F1 recipients carrying mutations at H-2K and/or I-A.
Open the PDF for Allosuppressor and allohelper T cells in acute and chronic graft-vs.-host disease. II. F1 recipients carrying mutations at H-2K and/or I-A. in another window
Journal Articles

Diseases caused by reactions of T lymphocytes towards incompatible structures of the major histocompatibility complex. VI. Autoantibodies characteristic of systemic lupus erythematosus induced by abnormal T-B cell cooperation across I-E.

F M van Rappard-van der Veen, A G Rolink, E Gleichmann
Journal: Journal of Experimental Medicine
J Exp Med (1982) 155 (5): 1555–1560.
https://doi.org/10.1084/jem.155.5.1555
Published: 01 May 1982
View article titled, Diseases caused by reactions of T lymphocytes towards incompatible structures of the major histocompatibility complex. VI. Autoantibodies characteristic of systemic lupus erythematosus induced by abnormal T-B cell cooperation across I-E.
Open the PDF for Diseases caused by reactions of T lymphocytes towards incompatible structures of the major histocompatibility complex. VI. Autoantibodies characteristic of systemic lupus erythematosus induced by abnormal T-B cell cooperation across I-E. in another window
Journal Articles

Allosuppressor and allohelper T cells in acute and chronic graft-vs-host disease. I. Alloreactive suppressor cells rather than killer T cells appear to be the decisive effector cells in lethal graft-vs.-host disease.

A G Rolink, T Radaszkiewicz, S T Pals, W G van der Meer, E Gleichmann
Journal: Journal of Experimental Medicine
J Exp Med (1982) 155 (5): 1501–1522.
https://doi.org/10.1084/jem.155.5.1501
Published: 01 May 1982
View article titled, Allosuppressor and allohelper T cells in acute and chronic graft-vs-host disease. I. Alloreactive suppressor cells rather than killer T cells appear to be the decisive effector cells in lethal graft-vs.-host disease.
Open the PDF for Allosuppressor and allohelper T cells in acute and chronic graft-vs-host disease. I. Alloreactive suppressor cells rather than killer T cells appear to be the decisive effector cells in lethal graft-vs.-host disease. in another window
Journal Articles

Capacity of genetically different T lymphocytes to induce lethal graft-versus-host disease correlates with their capacity to generate suppression but not with their capacity to generate anti-F1 killer cells. A non-H-2 locus determines the inability to induce lethal graft-versus-host disease.

E H Van Elven, A G Rolink, F V Veen, E Gleichmann
Journal: Journal of Experimental Medicine
J Exp Med (1981) 153 (6): 1474–1488.
https://doi.org/10.1084/jem.153.6.1474
Published: 01 June 1981
View article titled, Capacity of genetically different T lymphocytes to induce lethal graft-versus-host disease correlates with their capacity to generate suppression but not with their capacity to generate anti-F1 killer cells. A non-H-2 locus determines the inability to induce lethal graft-versus-host disease.
Open the PDF for Capacity of genetically different T lymphocytes to induce lethal graft-versus-host disease correlates with their capacity to generate suppression but not with their capacity to generate anti-F1 killer cells. A non-H-2 locus determines the inability to induce lethal graft-versus-host disease. in another window