The authors regret a factual error in the Fig. 7 legend. The corrected legend and Fig. 7 appear below.
Ovarian antigen exposure in neonatal mice capacitates AOD-specific T reg function. In this experiment, each neonatal recipient received 0.3 million CD4+CD25+ cells from the LNs of normal adult male or female mice. (A) AOD does not develop in the d3tx/nOX recipients of female T regs, whereas the d3tx/nOX recipients of male T regs developed AOD with the same incidence and severity as the AOD in control d3tx mice. (B) In contrast to AOD, dacryoadenitis in these d3tx/nOX recipients is inhibited completely by male or female T regs. (C) The ovarian graft of d3tx/nOX recipients of female T regs is normal histologically (arrow points to normal oocyte). (D) The ovarian graft of d3tx/nOX recipients of male T regs is infiltrated heavily by inflammatory cells, some replacing the oocyte (arrow; hematoxylin and eosin).
Ovarian antigen exposure in neonatal mice capacitates AOD-specific T reg function. In this experiment, each neonatal recipient received 0.3 million CD4+CD25+ cells from the LNs of normal adult male or female mice. (A) AOD does not develop in the d3tx/nOX recipients of female T regs, whereas the d3tx/nOX recipients of male T regs developed AOD with the same incidence and severity as the AOD in control d3tx mice. (B) In contrast to AOD, dacryoadenitis in these d3tx/nOX recipients is inhibited completely by male or female T regs. (C) The ovarian graft of d3tx/nOX recipients of female T regs is normal histologically (arrow points to normal oocyte). (D) The ovarian graft of d3tx/nOX recipients of male T regs is infiltrated heavily by inflammatory cells, some replacing the oocyte (arrow; hematoxylin and eosin).
