Two mouse urokinase-type plasminogen activator receptor (muPAR) cDNAs were isolated: muPAR1 is homologous to the human urokinase-type plasminogen activator receptor while muPAR2 codes for a 199 residue protein sharing the first 133 residues with muPAR1. Mouse genomic DNA sequencing indicates that the two different mRNAs arise by alternative splicing. In situ hybridization showed differential expression of the two mRNAs in mouse gastric mucosa. muPAR1 mRNA is located in luminal epithelial cells situated close to urokinase-type plasminogen activator-producing connective tissue cells of the lamina propria, pointing to plasmin generation controlled by the cooperation of different cells that may play a role in the release of gastric epithelial cells. muPAR2 mRNA is expressed in the basal epithelial cells, and the deduced protein sequence includes the receptor ligand binding domain, but omits the region involved in glycolipid-mediated membrane anchoring, suggesting that muPAR2 may code for a secreted uPA binding protein.
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15 December 1991
Article|
December 15 1991
Two alternatively spliced mouse urokinase receptor mRNAs with different histological localization in the gastrointestinal tract.
P Kristensen,
P Kristensen
Finsen Laboratory, Rigshospitalet, Copenhagen, Denmark.
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J Eriksen,
J Eriksen
Finsen Laboratory, Rigshospitalet, Copenhagen, Denmark.
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F Blasi,
F Blasi
Finsen Laboratory, Rigshospitalet, Copenhagen, Denmark.
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K Danø
K Danø
Finsen Laboratory, Rigshospitalet, Copenhagen, Denmark.
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P Kristensen
Finsen Laboratory, Rigshospitalet, Copenhagen, Denmark.
J Eriksen
Finsen Laboratory, Rigshospitalet, Copenhagen, Denmark.
F Blasi
Finsen Laboratory, Rigshospitalet, Copenhagen, Denmark.
K Danø
Finsen Laboratory, Rigshospitalet, Copenhagen, Denmark.
Online ISSN: 1540-8140
Print ISSN: 0021-9525
J Cell Biol (1991) 115 (6): 1763–1771.
Citation
P Kristensen, J Eriksen, F Blasi, K Danø; Two alternatively spliced mouse urokinase receptor mRNAs with different histological localization in the gastrointestinal tract.. J Cell Biol 15 December 1991; 115 (6): 1763–1771. doi: https://doi.org/10.1083/jcb.115.6.1763
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